1Barzilay JI, Abraham L, Heckbert SR, et al. The relation of markers of inflammation to the development of glucose disorders in the elderly: the Cardiovascular Health Study[J].Diabetes, 2001,50(10) : 2384-2389.
2Pradan AD, Manson JE, Rifai N, et al. C-reactive protein,Interleukin 6, and risk of developing type 2 diabetes mellitus[J]. JAMA, 2001,286(3) :327-334.
3Festa A, ErAgostino RJ, Howard G, et al. Chronic subclinical inflammation as part of the insulin resistance syndrome: the Insulin Resistance Atheroselerosis Study (IRAS) [J].Circulation, 2000,102 (1) : 42-47.
4Ford E. Body mass index, diabetes, and C-reactive protein among U. S. adults [J]. Diabetes Care, 1999,22 (12) : 1971-1977.
5Visser M, Bouter LM, Mc Quillan GM, et al. Elevated C-reactive protein levels in overweight and obese adults [J].JAMA, 1999, 282(22):2131-2135.
6Torzewski J, Torzewski M, Bowyer DE, et al. C-reactive protein frequently colocalizes with the terminal complement complex in the intima of early atherosclerotic lesions of human coronary arteries [J]. Arteriscler Thromb Vasc Biol, 1998,18(9) :1386-1392.
7Ridker PM, Cushman M, Stampfer MJ, et al. Inflammation,aspirin and risk of cardiovascular disease in apparently health men [J]. N Engl J Med, 1997,336(14) :973-979.
8Limb GA, Webster L, Soomro H, et al. Platalet expression of tumor necrosis factor-alpha (TNF-alpha), TNF receptor and intercellular adhesion molecule-1 (ICAM-1) in patients with proliferative diabetic retinopathy [J]. Clin Exp Immunool,1999,118(2) ,213-218.
9Zaremba J. Contribution of tumor necrosis factor alopha to the pathogensis of stroke [J]. Folia Morphol (Warsz), 2000, 59(3) :137-143.
10Schalkwijk CG, Poland DC, VanDijk W, et al. Plasma concentration of C-reactive protein is increased in type 1 diabetic patients without clinical macroangiopathy and correlates with markers of endothelial dysfunction., evidence for chronic inflammation [J]. Diabetologia, 1999,42(3):351-357.
二级参考文献8
1Cabay C, Kushner I. Acute-phase proteins and other systemic responses to inflammation. N Engl J Med, 1999,340:448-454.
2Donald E. Increased levels of acute-phase serum proteins in diabetes. Metabolism, 1989,38:1042-1046.
3Pickup JC, Crook MA. Is type 2 diabetes mellitus a disease of the innate immune system. Diabetologia, 1998,41:1241-1248.
4Martin A, Crook P, John C, et al. Relationship between plasma sialic acid concentration and microvascular and macrovascular complications in type 1 diabetes. Diabetes Care, 2001,24:316-321.
5Kunsch C, Medford RM. Oxidative stress as a regulator of gene expression in the vasculature. Circ Res, 1999,85:753-766.
6Erbagei A, Tarakcioglu M, Coskum Y, et al. Mediators of inflammation in children with type 1 diabetes mellitus: cytokines in type 1 diabetic children. Clin Biochem, 2001,34:645-650.
7Pickup JC, Mattock MB, Chusney GD, et al. NIDDM as disease of the innate immune system: association of acute reactants and interleukin-6 with metabolic syndrome X. Diabetologia, 1997,40:1286-1292.
8. Observations Subclinical inflammation in newly detected Type II diabetes and impaired glucose tolerance[J] 2002,Diabetologia(1):151
