期刊文献+

维持性血液透析患者骨保护蛋白及核因子κB受体活化子配体的研究 被引量:2

原文传递
导出
摘要 肾性骨病是维持性透析患者最常见的并发症之一。既往研究认为,肾性骨病的发病机制主要与继发性甲状旁腺功能亢进、1,25-(OH)2D,不足、钙磷代谢紊乱、酸中毒及铝中毒等因素有关,但临床上肾性骨病的发病并不能完全用上述因素来解释。
出处 《中华肾脏病杂志》 CAS CSCD 北大核心 2006年第3期170-171,共2页 Chinese Journal of Nephrology
基金 广东省自然科学基金(020426) 广东省重点攻关项目科研基金(2004B30701006) 广州市重点攻关项目(2005Z3-E0121) 广东省卫生厅基金(A2005036)
  • 相关文献

参考文献7

  • 1Simonet WS,Lacey DL,Dunstan CR,et al.Osteoprotegerin:a novel secreted protein involved in the regulation of bone density.Cell,1997,89:309-319.
  • 2Hsu H,Lacey DL,Dunstan CR ,et al.Tumor necrosis factor receptor family member RANK mediates osteoclast differentiation and activation induced by osteoprotegerin ligand.Proc Natl Acad Sci USA,1999,96:3540-3545.
  • 3Coen G,Ballanti P,Balducci A,et al.Serum osteoprotegerin and renal osteodystrophy.Nephrol Dial Transplant,2002,17:233-238.
  • 4许多荣,余学清,方荸荠.甲状旁腺激素在体外对破骨细胞分化及骨重吸收能力的影响[J].中华肾脏病杂志,2005,21(4):186-190. 被引量:14
  • 5Pei Y,Hercz G,Greenwood C,et al.Renal osteodystrophy in diabetic patients.Kidney Int,1993,44:159-164.
  • 6Avbersek LI,Balon BP,Rus I,et al.Increased bone resorption in HD patients:is it caused by elevated RANKL synthesis?Nephrol Dial Transplant,2005,20:566-570.
  • 7Huang JC,Sakata T,Pfelger LL,et al.iPTH differentially regulates expression of RANKL and OPG.J Bone Miner Res,2004,19:235-244.

二级参考文献14

  • 1Fong AM, Erickson HP, Zachariah JP, et al. Ultrastructure and function of the fractalkine mucin domain in CX3C chemokine domain presentation. J Biol Chem, 2000,75: 3781-3786.
  • 2Suda T, Takahashi N, Udagawa N, et al. Modulation of osteoclast differentiation and function by the members of the tumor necrosis factor receptor and ligand fanilies. Endocr Res, 1999,20: 345-357.
  • 3Duorong Xu,Zhenqi Shi,Xu Feng,et al. Development of a chimacric receptor approach to study signaling by tumour necrosis factor family members. Biochem J, 2004,383:219-225.
  • 4Wei L, Duorong X, Hongmei Y, et al. Functional identification of three RANK cytoplasmic motif mediating osteoclast differentiation and function. J Biol Chem, 2004,279:54759-54769.
  • 5Yasuda H, Shima N, Nakagawa N, et al. Osteoclast differentiation factor is a ligand for osteoprotegerin/osteogenesisinhibitory factor and is identical to TRANCE/RANKL.Proc Natl Acad Sci UAS, 1998,95:3597-3602.
  • 6Suda T, Takahashi N, Udagawa N, et al. Modulation of osteoclast differentiation and function by the members of the tumor necrosis factor receptor and ligand families. Endocr Res, 1999,20: 345-357.
  • 7Hofbauer L, Khosla S, Dunstan C, et al. The roles of osteoprotegerin and osteoprogerin ligand in the paracrine regulation of bone resorption. J Bone Miner Res,2000, 15:2-12.
  • 8Shi W, Michael WH, Steven L, et al. Interleukin-4 reversibly inhibits osteoclastogenesis via inhibition of NF-κB and Mitogen-activated protein kinase signaling. J Biol Chem,2002,277: 6622-6630.
  • 9Simonet W, Lacey D, Dunstan C, et al. Osteoprotegerin: A novel secreted protein involved in the regulation of bone density. Cell, 1997,89: 309-319.
  • 10Schiller P, Ippolito G, Roos B, et al. Anabolic or catabolic responses of MC3T3-E1 osteoblastic cells to parathyroid hormone depend on time and duration of treatment.J Bone Miner Res, 1999,14: 1504-1512.

共引文献13

同被引文献6

相关作者

内容加载中请稍等...

相关机构

内容加载中请稍等...

相关主题

内容加载中请稍等...

浏览历史

内容加载中请稍等...
;
使用帮助 返回顶部