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肺癌多阶段发病过程中的分子机制简述 被引量:3

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出处 《中国肺癌杂志》 CAS 2006年第2期211-214,共4页 Chinese Journal of Lung Cancer
基金 国家科技攻关计划项目(2002BA711A06) 北京科技计划重大项目(H0202220020510)资助
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参考文献44

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同被引文献23

  • 1董玉娥,沈建根,孟群,潘卫利,朱永良.人乳头瘤病毒和磷酸酪氨酸,SHP-2在尖锐湿疣组织重构中的表达研究[J].中国皮肤性病学杂志,2005,19(10):583-585. 被引量:1
  • 2唐蜜,李代强.磷酸化酪氨酸在非小细胞肺癌中的表达及其意义[J].实用预防医学,2007,14(1):30-32. 被引量:1
  • 3Jett J R,Midthun D E.Screening for lung cancer:current status and future directions:Thomas A.Neff lecture[J].Chest,2004,125(5S):158-62.
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  • 5Takahashi T,Nau M M,Chiba I,et al.p53:a frequent target for genetic abnormalities in lung cancer[J].Science,1989,246:491-4.
  • 6Gazzeri S,Gouyer V,Vour'ch C,et al.Mechanisms of p16INK4a inactivation in non small-cell lung cancers[J].Oncogene,1998,16(4):497-504.
  • 7Jin Y,Mandahl N,Heim S,et al.Isochromosomes i(8q) or i(9q) in three adenocarcinomas of the lung[J].Cancer Genet Cytogenet,1988,33(1):11-7.
  • 8Fan Y S,Li P.Cytog eneic studies of four human lung adenocarcinomas cell lines[J].Cancer Genet Cytogenet,1987,26(2):317-25.
  • 9Lee J,Pathak S,Hopwood V,et al.Involvement of chromosome 7 in primary lung tumor and nonmalignant normal lung tissue[J].Cancer Res,1987,47(23):6349-52.
  • 10Kitamira H,Kameda Y,Nakamura N,et al.Proliferative potential and p53 overexpression in precursor and early stage lesions of bronchioloalveolar lung carcinoma[J].Am J Pathol,1995,146(4):876-87.

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