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肾综合征出血热患者淋巴细胞亚群变化及其意义 被引量:1

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出处 《临床荟萃》 CAS 北大核心 2006年第10期730-731,共2页 Clinical Focus
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参考文献8

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  • 2王洪霞,徐建国.汉坦病毒致病机理的研究进展[J].疾病监测,2004,19(2):69-73. 被引量:1
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二级参考文献54

  • 1姚楚铮,黄莉莉,张满新,卢文红,刁保卫.肾综合征出血热病毒感染小鼠的抗体介导早死现象[J].中华微生物学和免疫学杂志,1994,14(1):16-18. 被引量:13
  • 2姚楚铮,黄莉莉.肾综合征出血热病毒感染小鼠的抗体介导早死的进一步研究[J].中华微生物学和免疫学杂志,1995,15(5):301-303. 被引量:8
  • 3董良全,王明照,窦金华,厉宣志,徐洪岚.内皮素在肾综合征出血热发病中的作用及其可能机制研究[J].中华传染病杂志,1996,14(4):228-229. 被引量:18
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  • 5Verheul H,Joma A, Hoekman K, et al. Vascular endothelial growth factor-stimulated endothelial cells promote adhesion and activation of platelets. Blood,2000,96: 4216-4221.
  • 6Sundstrom JB, McMullan LK, Spiropoulou CF, et al. Hantavirus infection induces the expression of RANTES and IP-10 without causing increased permeability in human lung microvascular endothelial cells. J Virol, 2001,75: 6070-6101.
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