摘要
hepatocyte，肝的主要细胞的部件，到损害的不同类型的展览变量危险性由内长或外长的因素导致了。肝细胞机能障碍或死亡和新生依赖于在众多的生物学上活跃的分子之间的复杂相互作用。激活血小板的因素(PAF ) 似乎在临床、试验性的背景作为肝损伤的关键调停人起一个枢轴的作用，由它的受体对手的有益的效果暗示了。处于与肝损伤联系的条件的 PAF 的特定的功能、规章的性质的全面新概述在这评论被尝试。
The hepatocyte, the main cellular component of the liver, exhibits variable susceptibility to different types of injury induced by endogenous or exogenous factors. Hepatocellular dysfunction or death and regeneration are dependent upon the complicated interactions between numerous biologically active molecules. Platelet- activating factor （PAF） seems to play a pivotal role as the key mediator of liver injury in the clinical and experimental setting, as implied by the beneficial effects of its receptor antagonists. A comprehensive up-to-date overview of the specific functional and regulatory properties of PAF in conditions associated with liver injury is attempted in this review.
