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Q-T间期延长中的室性早搏二联律伴早搏电交替

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出处 《中国医师进修杂志(内科版)》 2006年第7期60-61,共2页 Chinese Journal of Postgraduates of Medicine
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参考文献7

  • 1裘中,黄尚健.室性早搏二联律伴早搏电交替1例[J].湖南医科大学学报,2002,27(3):258-258. 被引量:1
  • 2Molnar J,Zhang F, Wells J, et al. Diurnal pattern of QTc interval:how long is prolonged. JACC, 1996, 27(1):76 -83.
  • 3Dekker JM, Crow RS, Hannah PJ, et al. Heart rate-corrected QT interval prolongation predicts risk of coronary heart disease in black and white middle-aged men and women. JACC, 2004,43(4):565- 571.
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  • 5刘金秋,杨延宗,Kenneth R Laurita,David S Rosenbaum.长QT综合征复极化离散与早期后除极致心律失常机制的跨壁光学标测研究[J].中华心律失常学杂志,2004,8(4):236-241. 被引量:6
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二级参考文献14

  • 1Girouard SD, Pastore JM, Laurita KR, et al. Optical mapping in a new guinea pig model of ventricular tachycardia reveals mechanisms for multiple wavelengths in a single reentrant circuit. Circulation, 1996, 93: 603-613.
  • 2Shimizu W, Antzelevitch C. Sodium channel block with mexiletine is effective in reducing dispersion of repolarization and preventing torsade de pointes in LQT2 and LQT3models of the long-QT syndrome. Circulation, 1997, 96:2038-2047.
  • 3Studenik CR, Zhou ZF, January CT. Differences in action potential and early afterdepolarization properties in LQT2 and LQT3 models of long QT syndrome. Br J Pharmacol, 2001 ,132: 85-92.
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  • 7Akar FG, Yan GX, Antzelevitch C, et al. Unique topographical distribution of m cells underlies reentrant mechanism of torsade de pointes in the long-QT syndrome. Circulation, 2002, 105: 1247-1253.
  • 8Antzelevitch C, Sicouri S, Litovsky SH, et al. Heterogeneity within the ventricular wall: electrophysiology and pharmacology of epicardial, endocardial and M cells. Circ Res, 1991, 69: 1427-1449.
  • 9Antzelevitch C, Shimizu W, Yan GX, et al. The M cell:its contribution to the ECG and to normal and abnormal electrical function of the heart. J Cardiovasc Electrophysiol,1999, 10: 1124-1152.
  • 10Moss AJ. Long QT syndrome. JAMA, 2003, 289: 2041-2044.

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