摘要
目的:探讨屈光参差性弱视和屈光不正性弱视的发病机理。方法:用幼猫视觉发育期单眼和双眼慢性阿托品化的方法塑造上述两种动物模型,成年后摘除外膝体,进行GolgiCox染色,观察计算背侧外膝体(dorsallateralgeniculatenucleus,dLGN)A1层的胞体截面积的树突野的有关参数。结果:实验动物dLGNA1层的class1和class2细胞胞体的截面积和树突野各参数的改变均为:单眼阿托品化眼所支配的细胞与正常眼的差异均有显著性,双眼阿托品化眼与正常眼差异有显著性,单眼阿托品化眼与双眼阿托品化眼之间差异无显著性。结论:屈光参差性弱视与屈光不正性弱视可能具有相似的发病机制。
PURPOSE:Toexplorethepathogenesisofanisometropicandametropicamblyopias.METHODS:Tocarryoutonmonocularandbinocularatropinizedcatmodelsduringthedevelopmen-talperiodforanisometropiaandametropia,andmeasurethecytosomalsectionalareaandsomepa-rametersofthedendricfieldfromthedorsallateralgeniculatenuclei(dLGN)ofadultcatsbyusingGolgi-Coxstaining.RESULTS:Thechangesofcytosomalsectionalareasandparametersaboutden-dricfieldsinthedLGNofexperimentalcatswereasfolowing:significantdiferencesbetweencelsofdLGNsA1laminabythemonocularatropinizedeyesandnormalones,binocularatropinizedeyesandnormalones;nosignificantdifferencebetweenthatdrivenbythemonocularandbinocularat-ropinizedeyes.CONCLUSIONS:Theremightberesemblepathogenesisbetweenanisometropicandametropicamblyopias.
出处
《中华眼底病杂志》
CAS
CSCD
1996年第3期153-156,共4页
Chinese Journal of Ocular Fundus Diseases
关键词
阿托品化
弱视
病理生理学
外膝体
动物模型
AtropineDiseaseModel,AnimalAmblyopia/physiopathologyCatsLateralgeniculatebody