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p38信号通路激活与肾小球硬化的研究进展

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摘要 肾小球硬化是各种肾小球疾病发展的最终结局，是慢性。肾小球肾炎进展的主要原因。细胞内信号通路在肾小球硬化中的作用是目前研究的热点。MAPK（有丝分裂原激活的蛋白激酶）是调节细胞内一系列病理、生理功能改变的重要信号通路，p38MAPK通路在炎症、应激、细胞周期、凋亡等多种细胞生理／病理过程发挥着重要的作用，本文综述如下。
作者 李清初 谷翠芝 尹友生
机构地区 桂林医学院附属医院肾内科
出处 《临床肾脏病杂志》 2006年第4期188-189,共2页 Journal Of Clinical Nephrology
关键词 有丝分裂原激活的蛋白激酶 P38信号通路 肾小球硬化 细胞内信号通路 P38MAPK 病理过程 细胞生理 肾小球肾炎 疾病发展 功能改变
分类号 R256.3 [医药卫生—中医内科学] R364.5 [医药卫生—病理学]
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参考文献14

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二级参考文献23

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临床肾脏病杂志
2006年 第4期

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