摘要
To the Editor： We are pleased to respond to the letter of Zarogiannis and his colleagues for their interest in our study, emphasizing the role of human visceral pleura in pleura fluid turnover. We definitely agree to Zarogiannis and colleagues＇ comments that the absence of many cuboidal cells in the visceral pleura does not favor the existence of visceral lymphatic stomata. However, any medical basic theory must be tested clinically, or used to interpret clinical phenomena well. The current model of pleural fluid turnover is hard to explain more frequently those with pleural effusion found in patients with merely left heart failure than those with merely right heart failure.
To the Editor： We are pleased to respond to the letter of Zarogiannis and his colleagues for their interest in our study, emphasizing the role of human visceral pleura in pleura fluid turnover. We definitely agree to Zarogiannis and colleagues＇ comments that the absence of many cuboidal cells in the visceral pleura does not favor the existence of visceral lymphatic stomata. However, any medical basic theory must be tested clinically, or used to interpret clinical phenomena well. The current model of pleural fluid turnover is hard to explain more frequently those with pleural effusion found in patients with merely left heart failure than those with merely right heart failure.
