2Dazzi M A,Chowdri N A,Kaul S K,et al. Evaluation of various methods of treating keloids and hypetrophic scars:a 10-year follow-up study[J]. Br J Plast Surg, 1992,45(2) : 374-379.
3Wagler W, Alfrink M, Micke O, et al. Results of prophylactic irradiation in patients with resected keloids:a retrospective analysis[J]. Acta Oncol, 2000,39(1) : 217-220.
4Klumpar D I, merray J C, Anscher M. keloids treated with excision followed by radiation therapy[J]. J Am Acad Dermatol, 1994,31(2) : 225 -231.
5Chaudhry M R, Alktar S, Duvalsaint F, et al. Ear lobe keloids,surgical exicision followed by radiation therapy: a 10-year experience[J]. Ear Nose Throat J, 1994, 73(5) : 779-781.
6Ship A G, Weiss P R, Mincer F R, et al. Sternal keloids:successful treatment employing surgery and adjunctive irradiation [J]. Ann Plast Surg, 1993, 31 (3) : 481-487.
7Sclafani A P, Gordon L, Chadha M, et al. Prevention of ear lobe keloid recurrence with postoperative corticosteroid injections Venus radiation therapy:a randomized,prospective study and review of the literature[J]. Dermatol Surg,1996,22(4):569-574.
8Ogawa R, Mitsuhashi K, Hyakusoku H, et al. Postoperative electron beam irradiation therapy for keloid[J]. Plast Reconstr Surg,2002,22(2) : 357-361.
9Han C M, Ishikura H, Tsukada S. Uhrastructure changes following electron irradiation in three-dimensional culture of normal human dermal fibroblasts[J]. Japanese J Plast Reconstr Surg,1994,14(1):89-97.
二级参考文献32
1Ueda,K.,Furuya,E.,Yasuda,Y.,et al.keloid have continuous high metabolic activity.Plast.Reconstr.Surg,1999,104:694-698.
2Tuan,T.L.,Nichter,L.S.The molecular basis of keloid and hypertrophic scar formation.Mol.Med.Today,1998,4:19-24.
3Greenhalgh,D.G.The role of apoptosis in wound healing.Int.J.Biochem.Cell.Biol,1998,30:1019-1030.
4Desmouliere,A.,Redard,M.,Darby,l.,et al.Apoptosis mediates the decrease in cellularity during the transition between granulation tissue and scar.Am.J.of Pathol,1995,146:56-60.
5Appleton,L.,Brown,N.J.,Willoughby,D.A.Apoptosis,Necrosis,and Proliferation possible implications in the etiology of keloid.Am.J.of Pathol,1996,149:1441-1450.
6Saed,G.M.,Ladin,D.,Olson,J.,et al.Analysis of p53 gene mutation in keloids using polymerase chain reaction-based single-strand conformational polymorphism and DNA sequencing.Arch.Dermatol,1998,134:963-972.
7Teofoli,P.,Barduagni,S.,Ribuffo,M.,et al.Expression of Bcl-2,p53 ,c-jun and c-fos protooncogenes in keloids and hypertrophic scars.J.Dermatol.Sci,1999,22:31-35.
8Akasaka,Y.,Ishikawa,Y.,Ono,I.,et al.Enhanced expression of caspase-3 in hypertrophic scar and keloid:Induction of caspase-3 and apoptosis in keloid fibroblasts in vitro.Lab.Invest,2000,80:345-355.
10Armstrong,RC.,Aja,T.,Xiang,J.,et al.Fas-induced activation of the cell deathrelated protease CPP32 is inhibited by Bcl-2 and by ICE family protease inhibitors.J.Biol.Chem,1996,271:16850-16858.
8Joachim Kutzner,Lida Schneider,Michael Heinrich Seegenschmiedt.Strahlentherapie des Keloids in Deutschland Patterns-of-Care-Studie – Ergebnisse einer Umfrage[J]Strahlentherapie und Onkologie,2003(1).
9Luo S,Benathan M,Raffoul W,et al.Abnormal balance between proliferation and apoptotic cell death in fibroblasts derived from keloid lesionsPlastic and Reconstructive Surgery,2001.
10Guix B,Henriquez I,Andres A,et al.Treatment of keloids by high-dose-rate brachytherapy: A seven-year studyInternational Journal of Radiation Oncology Biology Physics,2001.
