3[1]Louw DF, Masada T, Sutherland GR. Ischemic neuronal injury is ameliorated by astrocyte activation. Can J Neurol Sci, 1998, 25: 102
4[2]Kirino T, Sano K. Selective vulnerability in the gerbil hippocampus followin g transient ischemia. Acta Neuropathol Berl, 1984, 62: 201
5[3]Lin B, Ginsberg MD, Busto R, et al. Sequential analysis of subacute and chron ic neuronal, astrocytic and microglial alterations after transient global ischem ia in rats. Acta Neuropathol Berl, 1998, 95: 511
6[4]Perez-Pinzon MA, Mumford PL, Rosenthal M, et al. Anoxic preconditioning in hippocampal slices: role of adenosine. Neurosci, 1996, 75:687
7[5]Perez-Pinzon MA, Xu GP, Dietrich WD, et al. Rapid preconditioning protects r ats against ischemic neuronal damage after 3 but not 7 days of reperfusion follo wing global cerebral ischemia. J Cereb Blood Flow Metab, 1997, 17: 175
8[6]Lascola C, Kraig RP. Astroglial acid-base dynamics in hyperglycemic and norm oglycemic global ischemia. Neurosci Biobehav Rev, 1997, 21: 143
9[7]Schurr A, Payne RS, Miller JJ, et al. Glia are the main source of lactate utiliz ed by neurons for recovery of function posthypoxia. Brain Res, 1997, 774: 221
10[8]Westenbroek RE, Bausch SB, Lin RC, et al. Upregulation of L-type Ca2+ cha nnels in reactive astrocytes after brain injury, hypomyelination, and ischemia. J Neurosci,1998, 18: 2321
