1Sims AM,Wordsworth BP,Brown MA.Genetic susceptibility to ankylosing spondylitis.Curr Mol Med.2004,4(1):13.
2Goodall Jane C,Ellis Lou,Hill Gaston JS.Spondyloarthritis-associated B27 subtypes differ in their dependence upon tapasin for surface exepression and their incorporation into the peptide loading complex.Arthritis and Rheumatism.2006,54(1):138.
3Martin T,Fiorillo Monica Maragnc,et al.CD8+ T-cell autoreactivity to an HLA-B27-Restricted self-epitope correlates with AS.Clin Ivest.2000,106:47.
4Martin Hu Ismeyer,Maria Teresa,et al.Dual HLA-B27 Subtype-dependent comformation of a self-peptide.Exp Med.2004,199(2):271.
5John D,Reveille MD,Edward J,et al.HLA-B27 and genetic predisposing factors in spondyloarthropathies.Rheumatol.2001,13:265.
6Wei Jcc,Tsaiwe,et al.HLA-B60 and B61 are strongly associated with ankylosing spondylitis in HLA-B27-negative Taiwan Chinese patients.Rheumatol.2004,43(1):839.
7Chen CH,Ljn KC,Yu DT,et al.Serum matrix metalloproteinases and tissue inhibitors of metalloproteinases in ankylosing spondylitis:MMP-3 is a reproducibly sensitive and specific biomarker of disease activity.Rheumatol.2006,45(4):414.
8Jin L,Weisman M,Zhang G,et al.Lack of association of matrix metalloproteinase 3 (MMP3) genotypes with ankylosing spondylitis susceptibility and severity.Rheumatol.2005,44(1):55.
10Stone M A,Payne U,et al.Comparative immune response to candidate arthritogenic bacteria do not confirm a dominant role for klebsiella pneumonia in the pathogenesis of familial ankylosing spondylitis.Rheumatol.2004,43:148.
二级参考文献32
1[1]Brewerton DA,Caferry M,Hart FD,et al.Ankylosing spondylitis and HLA-B27[J].Lancet,1973,1(7809):904-907.
2[2]Braun J,van der Heijde D.Novel approaches in the treatment of ankylosing spondylitis and other spondyloarthritides[J].Expert Opin Investig Drugs,2003,12(7):1097-1109.
3[3]van der Linden S,Valkenburg HA,Cats A.Evaluation of diagnostic criteria for ankylosing spondylitis.A proposal for modification of the New York criteria[J].Arthritis Rheum,1984,27(4):361-368.
4[4]Braun J,Bollow M,Neure L,et al.Use of immunohistologic and in situ hybridization techniques in the examination of sacroiliac joint biopsy specimens from patients with ankylosing spondylitis[J].Arthritis Rheum,1995,38(4):499-505.
5[6]González S,Torre-Alonso JC,Martínez-Borra J,et al.TNF-238A promoter polymorphism contributes to susceptibility to ankylosing spondylitis in HLA-B27 negative patients[J].J Rheumatol,2001,28(6):1288-1293.
6[7]Milicic A,Lindheimer F,Laval S,et al.Interethnic studies of TNF polymorphisms confirm the likely presence of a second MHC susceptibility locus in ankylosing spondilitis[J].Genes Immun,2000,1(7):418-422.
7[10]Vinasco J,Beraún Y,Nieto A,et al.Polymorphism at the TNF loci in rheumatoid arthritis[J].Tissue Antigens,1997,49(1):74-78.
8Ikawa T,Ikeda M,Yamaguchi A et al.Expression of arthritis-causing HLA-B27 on Hela cells promotes induction of c-fos in response to in vitro invasion by Salmonella typhimurium [J].J Clin Invest,1998;101:263.
9Hermann E,Yu D,Meyer Zum Buschenfelde K et al.HLA-B27 restricted CD8+T cells derived from synovial fluids of patients with reactive arthritis and ankylosing spondylitis [J].Lancet,1993;342:646.
10Colbert R.HLA-B27 misfolding:a solution to the spondyloarthropathy conundrum? [J].Mol Med Today,2000;6:224.