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高血压早期肾损害相关因素研究及药物干预的影响 被引量:12

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出处 《临床荟萃》 CAS 北大核心 2007年第3期189-191,共3页 Clinical Focus
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  • 1Brenner BM. Mackenzie HS. Nephron mass as a risk factor for progression of renal disease[J], Kidney Int, 1997,63 (Suppl) : S124-S127.
  • 2Zoja C, Benigni A, Remuzzi G, et al. Cellular responses to protein overload:key event in renal disease progession[J]. Curr Opin Nephrol Hypertens,2004,13(1):31-37.
  • 3孙明,吴峻.两类尿蛋白对不同时期高血压肾损害的相关性研究[J].中国现代医学杂志,2002,12(16):10-13. 被引量:8
  • 4Palatini P,Graniero GR, Canali C, et al. Relationship between albumin excretion rate, ambulatory blood pressure and left ventricular hypertrophy in mild hypertension[J]. J Hypertens, 1995,13(12 Pt 2):1796-1800.
  • 5Chagnac A, Weinstein T, Korzets A, et al. Glomerular hemodynamics in severe obesity [J]. Am J Physiol Renal Physiol, 2000,278(5) :F817-822.
  • 6Kambham N,Markowitz GS,Valeri AM,et al. Obesity-related glomerulopathy : an emerging epidemic[J]. Kidney Int, 2001,59(4) : 1498-1509.
  • 7Weber M. The telmisartan programme of research to show telmisartan end-organ protection programme[J]. J Hyertens, 2003,21(Suppl 6) :S37-S46.
  • 8Benson SC,Pershadsingh HA, Ho CI, et al. Indentification of telmisartan as a unique angiotension Ⅱ receptor antangonist with selective PPAR gamma-modulating activity [ J ].Hypertension, 2004,43 (5) :993-1002.
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