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类风湿关节炎新生血管形成中细胞因子的作用

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作者 朱丽艳 杨林
出处 《华北煤炭医学院学报》 2007年第3期323-325,共3页 Journal of North China Coal Medical College
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参考文献20

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  • 2Lafyatis R,Remmers EF,Roberts A B,et al.Anchorage-independent growth of synoviocytes from arthritic and normaljoints.Stimulation by exogenous platelet-derived growth factor and inhibition by transforming growth factor-beta and retinoids[J].J Clin Invest,1989,83(4):1267
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  • 7Wakiaka S,Snznki S,Takclba Y,et al.MIodulnlation by proinflammatory cytokina,of Fay/Fas; ligand -mediated apoptosis cell death of svnovial cells with rhcnmatoid arthritis[J].Clin Exp Immunol,1998,114(1):119
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二级参考文献6

  • 1Yamanishi Y, Firestein GS. Pathogenesis of rheumatoid arthritis: the role of synoviocytes. Rheum Dis Clin Noi, th Am, 2001, 27(2): 355 - 371.
  • 2Koch AE. Review: angiogenesis: implications for rheumatoid arthritis.Arthritis rheumatism, 1998, 41 (6): 951 -965.
  • 3Li DY, Scrensen LK, Brooks BS, et al. Defective angiogenesis in mice lacking endoglin. Science, 1999, 284(5419): 1534- 1537.
  • 4Szekanecz Z, Haines GK, Harlow LA, et al. Increased synovial expression of TGF-β receptor endoglin and TGF-β1 in rheumatoid arthritis. Clin Immunol Immunopathol, 1995, 76(2): 187 - 194.
  • 5Levitt ML Koty PP. Tyrosine kinase inhibitors in preclinical development. Invest New Drugs, 1999, 17(3): 213 -216.
  • 6陈国庭,尹浩然,朱正刚.Endoglin:肿瘤新生血管的标志物[J].国外医学(生理病理科学与临床分册),2002,22(1):7-8. 被引量:7

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