1[1]Maltsev VA,Undrovinas AI.A multi-modal composition of the late Na+ current in human ventricular cardiomyocytes[J].Cardiovasc Res,2006,69(1):116-127.
2[2]Moric E,Herbert E,Trusz-Gluza M,et al.The implications of genetic mutations in the sodium channel gene (SCN5A)[J].Europace,2003,5(4):325-334.
3[3]Opie LH.Tedisamil in coronary disease:additional benefits in the therapy of atrial fibrillation?[J].J Cardiovasc Pharmacol Ther,2003,8 (Suppl 1):S33-S37.
4[4]Brown AM.Drugs,hERG and sudden death[J].Cell Calcium,2004,35(6):543-547.
5[5]Miake J,Marbán E,Nuss HB.Functional role of inward rectifier current in heart probed by Kir2.1 overexpression and dominant-negative suppression[J].J Clin Invest,2003,111(10):1529-1536.
6[6]Priori SG,Pandit SV,Rivolta I,et al.A novel form of short QT syndrome (SQT3) is caused by a mutateon in the KCNJ2 gene[J].Circ Res,2005,96 (7):800-807.
7[7]Kaab S,Zwermann L,Barth A,et al.Selective block of sarcolemmal IKATP in human cardiomyocytes using HMR1098[J].Cardiovasc Drugs Ther,2003,17(5 6):435-441.
8[8]Patterson E,Lazzara R,Szabo B,et al.Sodium-calcium exchange initiated by the Ca2 + transient:an arrhythmia trigger within pulmonary veins[J].J Am Coll Cardiol,2006,47(6):1196-1206.
9[9]Splawski I,Timothy KW,Sharpe LM,et al.Cav 1.2 calcium channel dysfunction causes a multisystem disorder including arrhythmia and autism[J].Cell,2004,119 (1):19-31.
10[10]Harris AL.Emerging issues of connexin channels:biophysics fills the gap[J].Q Rev Biophys,2001,34 (3):325-472.
5Ravingerova T, Tribulova N, Slezak J, et al. Brief,intermediate and prolonged ischemia in the isolated crystalloid perfused rat heart :relationship between susceptibility to arrhythmias and degree of ultrastructural injury[J]. J Mol Cell Cardiol, 1995,27 (9): 1937-1951.
6Puglisi J L,Bassani R A,Bassani J W, et al. Temperature and relative contributions of Ca transport systems in cardiac myocyte relaxation [J]. Am J Physiol,1996, 270 (5 Pt 2):H1772- H1778.
7Wu T J,Ong J J,Chang C M,et al. Pulmonary veins and ligament of Mashall as sources of rapid activations in canine model of sustained atrial fibrillation[J]. Circulation, 2001, 103 (8) :1157-1163.
8Arenzana-Seisdedos F, Turpin P, Rodriguez M,et al. Nuclear localization of I-κB promotes active transport of NF-κB from the nucleus to the cytoplasm[J]. J Cell Sci, 1997, 110 (Pt 3) :369-378.
9Nao T,Ohkusa T,Hisamatsu Y,et al. Comparison of expression of connexin in right atrial myocardium in patients with chronic atrial fibrillation versus those in sinus rhythm[J]. Am J Cardiol,2003,91(6) :678-683.
10Nao T,Ohkusa T,Hisamatsu Y,et a1.Comparison of expressionof connexin in righta trial myocardium in patients with chronicatrial fibrillation versus those in sinus rhythm[J].Am J Cardiol,2003,91(6):678-683.
