1[1]Chan FL, Choi HL, Chen ZY, et al. Induction of apoptosis in prostate cancer cell lines by a flavonoid, baicalin[J]. Cancer Lett,2000,160(2):219-228.
2[2]Werner S, Smole H. Paracrine regulation of keratinocyte proliferation and differention[J]. Trends Cell Biol,2001,11(4):143-146.
3[3]Oyama N, Iwatsuke K, Satoh M, et al. Dermal fibroblasts are one of the therapeutic targets for topical application of 1 alpha,25-dihydroxyvitamin D3: the possible involvement of transforming growth factor-beta induction[J]. Br J Dermatol,2000,143(6): 1140-1148.
4[4]Miura H, Sano S, Higashiyama M, et al. Involvement of insulinlike growth factor-1 in psoriasis as a paracrine growth factor: dermal fibroblasts play a regulatory role in developing psoriatic lesions[J]. Arch Dermatol Res,2000,292(12):590-597.
5[5]Ikezoe T, Chen SS, Heber D, et al. Baicalin is a major component of PC-SPES which inhibits the proliferation of human cancer cells via apoptosis and cell cycle arrest[J]. Prostate,2001,49(4):285-292.
6[6]Stein M, Bernd A, Ramirez-Bosca A, et al. Measurement of anti-inflammatory effects of glucocorticoids on human keratinocytes in vitro. Comparison of normal human kertinocytes with the keratinocyte cell line HaCaT[J]. Arzneimittelforschung, 1997,47:1266-1270.
7[7]Chen S, Ruan Q, Bedner E, et al. Effects of the flavonoid baicalin and its metabolite bacalein on androgen receptor expression, cell cycle progression and apoptosis of prostate cancer cell lines [J].Cell Prolif,2001,34(5):293-304.
8[8]Dimon-Gadal S, Raynaud F, Evain-Brion D, et al. MAP kinase abnormalities in hyperproliferative cultured fibroblasts from psoriatic skin[J]. J Invest Dermatol,1998,110 (6):872-879.