1Zannad F,Alla F,Dousset B,et al.Limitation of excessive extracellular matrix turnover may contribute to surviva benefit of spironolactone therapy in patients with congestive heart failure:insights from the randomized aldactone evaluation study (RALES)[J].Circulation 2000,102(22):2700-2706.
2Devereux RB,Reichek N.Echocardigraphic delemination of left ventricular mass in man; analomic validalion of tha method[J].Circulation,1997,55 (7):613.
3Sharon A,Hunt M.ACC/AHA Guidelines for the Evaluation and Management of Chronic Heart Failure in the Adult[J].J Am Coll Cardiol,2001,44(12):2021-2023.
4Dries DL,Stevenscen LW.Brain natriuretic peptid as bridge to therapy for hearrt failure[J].Lancet,2000,355 (9210):1112.
5Tsutamoto T,et al.Plasma BNP level as a biochemical marker of morbidity in pathients with asympotomatic or minimally sympotomatic[J].Eur Heart J,1999,20(8):1799-1806.
7Maisel A.B-type natriurwtic peptide levels:a potential novel white count for congetive heart failure[J].J Card Fail,2001,18(7):183-193.
8Eric J,Eichorn MD,Michael R,et al.Medical therapy can improve the biological properties of the chronically failying heart[J].Circulation,1996,194(10):2285-2296.
9Tsatsui H,Spinale FG,Nagatsu M,et al.Effects of chronic beta-adrenergic blockade on the left ventricular ang cardiocyte abnormalities of chronic canine mitral regurgitation[J].J Clin Invest,1994,93 (5):2639-2648.
10TAKEDA Y, YONEDA T, DEMURA M, et al. Effects of high sodium intake on cardiovascular aldosterone synthesis in stroke-prone spontaneously hypertensive rats[J]. Hypertens,2001,19(3) : 635-939.