1Sugita S, Khvochtev M, Sudhof TC. Neurexin are functional α-latrotoxin receptors[J]. Neuron, 1999, 22(3): 489-496.
2Rowen L, Young J, Birditt B, et al. Analysis of the human neurexin genes: alternative splicing and the generation of protein diversity [J]. Genomics, 2002, 79(4): 587-597.
3Tabuchi K, Sudhof TC. Structure and evolution of neurexin genes: insight into the mechanism of alternative splicing [J]. Genomics, 2002, 79(6): 849-859.
4Boucard AA, Chubykin AA, Comoletti D, et al. A splice code for trans-synaptic cell adhesion mediated by binding of neuroligin 1 to α- and β-Neurexins[J]. Neuron, 2005, 48(2): 229-236.
5Butz S, Okamoto M, Sudhof TC. Atripartite protein complex with the potential to couple synaptic vesicle exocytosis to cell adhesion in brain[J]. Cell, 1998, 94(6): 773-782.
6Song JY, Ichtchenko K, Stidhof TC, et al. Neuroligin 1 is a postsynaptic cell-adhesion molecule of excitatory synapses [J]. Proc Natl Acad Sci USA, 1999, 96(3): 1100-1105.
7Scheiffele P, Fan J, Choih J, et al. Neuroligin expressed in nonneuronal cells triggers presynaptic development in contacting axons[J]. Cell, 2000, 101 (6): 657-669.
8Fu Z, Washboume P, Ortinski P, et al. Functional excitatory synapses in HEK293 cells expressing neuroligin and glutamate receptors[J]. J Neurophysiol, 2003, 90(6): 3950-3957.
9Levinson JN, Chary N, Huang K, et al. Neuroligins mediate excitatory and inhibitory synapse formation[J]. J Biol Chem, 2005, 280(17): 17312-17319.
10Sara Y, Biederer T, Atasoy D, et al. Selective capability of SynCAM and neuroligin for functional synapse assembly [J]. J Neurosci, 2005, 25 (1): 260-270.
二级参考文献20
1Altrock WD, tom Dieck S, Sokolov M, et al. Functional inactivation of a fraction of excitatory synapses in mice deficient for the active zone protein Bassoon [J]. 2003, 37: 787-800.
2Rosahi TW, Ceppert M, Spillane D, et al. Short-term synaptic plasticity is altered in mice lacking synapsin I [J]. Cell, 1993, 75:661-670.
3Stevens CF. Neurotransmitter release at central synapses [J]. Neuron,2003, 40: 381-388.
4Srensen JB. Differential control of the releasable vesicle pools by SNAP-25 splice variants and SNAP-23 [J]. Cell, 2003, 114: 75-86.
6Bajjalieh SM. Synaptic vesicle docking and fusion [J]. Current Opinion in Neurobiology, 1999, 9: 321-328.
7Ungermann C, Sato K, Wickner W. Defining the functions of trans SNARE pairs [J]. Nature, 1998, 396: 543-548.
8Richmond JE, Broadie KS. The synaptic vesicle cycle: exocytosis and endocytosis in Drosophila and C. elegans [J]. Current Opinion in Neurobiology, 2002, 12: 499-507.
9Augustin I, Rosenmund C, Südhof TC, et al. Munc13-1 is essential for fusion competence of glutamatergic synaptic vesicles [J]. Nature,1999, 400: 457-461.
10Betz A, Thakur P, Junge HJ, et al. Functional interaction of the active zone proteins Muncl3-1 and RIM1 in synaptic vesicle priming [J]. ,2001, 30: 183-196.
4Ingram J L,Peckham S L,Tisdale B,et al.Prenatal exposure of rats to valproic acid reproduces the cerebellar anomalies assiated with autism.Neurotoxicol Teratol,2000,22(3):319-324.
5Kolozsi E,Mackenzie R N,Roullet F I.Exposure to Vampirical acid leads to reducedexpression of synaptic adhesion moleculeneuroligin-3 in mic e.Neuroscience,2009,163 (4) 1201 - 1210.
6Jamaica, S,Quach H,Betancur C,et al.Mutations of the X-linked genes encoding neuroligins NLGN3 and NLGN4 are associated with autism.Nat Genet,2003,34(1):27-29.
