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B淋巴细胞发育耐受异常在系统性红斑狼疮发病机制中的研究进展

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摘要 系统性红斑狼疮(systemic lupus erythatosus,SLE)是一种由于机体免疫系统功能异常而产生抗核抗体等多种自身抗体.形成的免疫复合物沉积而导致多器官多系统受累的自身免疫性疾病。
作者 曾婷 杨程德
出处 《中华风湿病学杂志》 CAS CSCD 2007年第12期747-750,共4页 Chinese Journal of Rheumatology
基金 国家自然科学基金资助项目(30772009) 上海市教委重点学科建设资助项目(T0203)
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参考文献28

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二级参考文献8

  • 1Gross JA,Johnston J,Mudri S,et al.TACI and BCMA are receptors for a TNF homologue implicated in B-cell autoimmune disease.Nature,2000,404:995-999.
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  • 3Ng LG,Sutherland AP,Newton R,et al.B cell-activating factor belonging to the TNF family (BAFF)-R is the principal BAFF receptor facilitating BAFF costimulation of circulating T and B cells.J Immunol,2004,173:807-817.
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  • 6Yu G,Boone T,Delaney J,et al.APRIL and TALL-I and receptors BCMA and TACI:system for regulating humoral immunity.Nat Immunol,2000,1:252-256.
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  • 8李东升,李家文.B淋巴细胞刺激物与系统性红斑狼疮的研究进展[J].国外医学(皮肤性病学分册),2003,29(5):291-293. 被引量:3

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