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骨关节炎软骨细胞凋亡的信号传导 被引量：1

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作者 高萌萌 张梅
机构地区 安徽医科大学附属省立医院
出处 《中国老年学杂志》 CAS CSCD 北大核心 2008年第2期200-202,共3页 Chinese Journal of Gerontology
基金 国家中医药管理局课题(2000-JP-58)
关键词 骨关节炎 软骨细胞 细胞凋亡 线粒体 内质网
分类号 R684.3 [医药卫生—骨科学]
  • 相关文献

参考文献23

  • 1于顺禄,李德达,李世民,刘长明,王毅,王介民.骨性关节炎研究进展[J].中国骨伤,2002,15(10):635-637. 被引量：49
  • 2Ruiz-Romero C, Lopez-Armada MJ, Blanco FJ. Mitochondfial proteomic characterization of human normal articular chondrocytes [ J]. Osteoarthritis Cartilaqe,2006; 14(6) :507-18
  • 3余小平.线粒体在细胞凋亡中的作用[J].国外医学：卫生学分册,2001,28(5):307-311.
  • 4Earnshaw WC, Martins LM, Kaufmann SH, et al. Mammalian caspases: structure, activation, substrates and functions during apoptosis [J]. Annu Rev Biochem, 1999 ;68:383-424.
  • 5Verhagen AM, Ekert PG, Pakusch M, et al. Identification of DIABLO, a mammalian protein that promotes aoplosis by binding to and antagonizingI IAP proteins[J]. Cell ,2000 ; 102( 1 ) :43-53.
  • 6Li K, Li Y, Shelton JM, et al. Cytochromic deficiency causes embryonic lethality and attenuates stress-induced apoptosis [ J]. Cell, 2000; 101 (4) :389-99.
  • 7Adrain C, Creagh EM, Martin SJ. Apoptosis-associated release of Smac/ DIABLO from mitochondria requires active caspases and is blocked by Bcl-2[J]. EMBO J,2001 ;20(23 ) :6627-36.
  • 8Pardo OE, Lesay A, Arcaro A, et al. Fibroblast growth factor 2-mediated translational control of IAPs blocks mitochondrial release of Smac / DIA- BLO and apoptosis in small cell lung cancer cells [J]. Mol Cell Biol, 2003 ;23 (21) :7600-10.
  • 9Vieira H, Kroemer G. Mitochondria as targets of apoptosis regulation by nitric oxide[J]. IUBMB Life,2003 ;55 ( 10-11 ) :613-20.
  • 10Reed JC ,Jurgensmeier JM, Matsuyama S. Bcl-2 family proteins and mitochondria[J]. Biochim Biophys Acts, 1998 ; 1366 (1-2) : 127-37.

二级参考文献45

  • 1Yin XM. Bid, a critical mediator for apoptosis induced by the activation of Fas/TNF-R1 death receptors in hepatocytes. J Mol Med, 2000, 78: 203-211.
  • 2Sartorius U, Schmitz I, Krammer PH. Molecular mechanisms of death-receptor-mediated apoptosis. Chembiochem, 2001, 2: 20-29.
  • 3Shimizu S, Konishi A, Kodama T, et al. BH4 domain of antiapoptotic Bcl-2 family members closes voltage-dependent anion channel and inhibits apoptotic mitochondrial changes and cell death. Proc Natl Acad Sci U S A, 2000, 97: 3100-3105.
  • 4Yajima H, Suzuki F. Identification of a Bcl-XL binding region within the ATPase domain of Apaf-1. Biochem Biophys Res Commun, 2003, 309: 520-527.
  • 5Degterev A, Lugovskoy A, Cardone M, et al. Identification of small-molecule inhibitors of interaction between the BH3 domain and Bcl-xL. Nat Cell Biol, 2001, 3: 173-182.
  • 6Schwabe RF, Brenner DA. Role of glycogen synthase kinase-3 in TNF-alpha-induced NF-kappaB activation and apoptosis in hepatocytes. Am J Physiol Gastrointest Liver Physiol, 2002, 283: G204-211.
  • 7Beutler B, Bazzoni F. TNF, apoptosis and autoimmunity: a common thread? Blood Cells Mol Dis, 1998, 24: 216-230.
  • 8Zheng SJ, Wang P, Tsabary G, et al. Critical roles of TRAIL in hepatic cell death and hepatic inflammation. J Clin Invest, 2004, 113: 58-64.
  • 9Kanzler S, Galle PR. Apoptosis and the liver. Semin Cancer Biol, 2000,10: 173-184.
  • 10Natoli G, Costanzo A, Guido F, et al. Nuclear factor kB-independent cytoprotective pathways originating at tumor necrosis factor receptor-associated factor 2. J Biol Chem, 1998, 273: 31262-31272.

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中国老年学杂志
2008年 第2期

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