6Gross J. Getting to mammalian wound repair and amphibian limb regeneration: a mechanistic link in the early events [ J ]. Wound Rep Reg, 1996,4 (2) : 190 - 202.
7Lekic PC, Pender N, Mcculloch CA. Is fibroblast heterogeneity relevant to the heath, diseases, and treatments of periodontal tissues[J] 9. Crit Rev Oral Biol Med,1997,8(3):253 - 268.
8Rinn JL, Bondre C, Gladstone HB, et al. Anatomic demarcation by positional variation in fibroblast gene expression programs [ J ]. PLoS Genet,2006,2 (7) 1084 - 1096.
9Sorrell JM, Baber MA, Caplan AI. Clonal characterization of fibroblasts in the superficial layer of the adult human dermis [ J ]. Cell Tissue Res,2007,327 (3) :499 - 510.
10Sorrell JM, Baber MA, Caplan AI. Site - matched papillary and reticular human dermal fibroblasts differ in their release of specific growth factors/cytokines and in their interaction with keratinocytes [ J ]. J Cell Physiol,2004,200 ( 1 ) : 134 - 145.
7Pages G, Lenormand P, L'Allemain G, et al.Mitogen-activated protein kinases p42mapk and p44mapk are required for fibroblast proliferation.Proc Natl Acad Sci U S A 1993:90(18):8319-23.
9Ihn H, Tamaki K. Oncostatin M stimulates the growth of dermal fibroblasts via a mitogen-activated protein kinase-dependent pathway. J Immunol 2000;165(4):2149-55.
10Brogley MA, Cruz M, Cheung HS. Basic calcium phosphate crystal induction of collagenase 1 and stromelysin expression is dependent on a p42/44 mitogenactivated protein kinase signal transduction pathway.J Cell Physiol 1999;180(2):215-24.
1Fries KM, Blieden T ,Looney RJ ,et al. Evidence of fibroblast heterogeneity and the role of fibroblast subpopulations in fibrosis [J]. Clin Immunollmmunopathol,1994,72(3) :283-292.
2Sorrell JM, Caplan AI. Fibroblast heterogeneity: more than skin deep [J]. J Cell Sci ,2004,117 (Pt 5) :667-675.
3Honardoust D, Varkey M, Hori K, et al. Small leucine-rich proteoglycans ,decorin and fibromodulin, are reduced in posthurn hypertrophic scar[J]. Wound Repair Regen ,2011 ,19 (3) :368-378.
4Wang J, Hori K, Ding J ,et al. Toll-like receptors expressed by dermal fibroblasts contribute to hypertrophic scarring[J]. J Cell Physiol,2011 ,226(5) :1265-1273.
5Wang J, Dodd C, Shankowsky HA, et al, Deep dermal fibroblasts contribute to hypertrophic scarring[J]. Lab Invest ,2008,88 (12) : 1278-1290.
6Varkey M, Ding J, Tredget EE. Differential collagen-glycosaminoglycan matrix remodeling by superficial and deep dermal fibroblasts: potential therapeutic targets for hypertrophic scar [J]. Biomaterials,2011 ,32(30) :7581-7591.
7Honardoust D, Ding J, Varkey M, et al. Deep dermal fibroblasts refractory to migration and decorin-induced apoptosis contribute to hypertrophic scarring [J]. J Bum Care Res ,2012 ,33 (5) :668-677.
8Supp DM, Hahn JM, Glaser K, et al. Deep and superficial keloid fibroblasts contribute differentially to tissue phenotype in a novel in vivo model of keloid scar[J]. Plast Reconstr Surg ,2012,129 (6) : 1259-1271.
9Seifert 0, Bayat A, Geffers R, et al. Identification of unique gene expression patterns within different lesional sites of keloids [J]. Wound Repair Regen ,2008 ,16(2) :254-265.
10Iqbal SA, Syed F, Mcgrouther DA, et al. Differential distribution of haematopoietic and nonhaematopoietic progenitor cells in intralesional and extralesional keloid: do keloid scars provide a niche for nonhaematopoietic mesenchymal stem cells? [J]. Br J Dermatol, 2010, 162( 6) : 1377-1383.
