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下丘脑小胶质细胞参与充血性心力衰竭的发病过程

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机构地区 山西医科大学
出处 《中西医结合心脑血管病杂志》 2008年第2期199-201,共3页 Chinese Journal of Integrative Medicine on Cardio-Cerebrovascular Disease
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参考文献31

  • 1Kang YM, Shao TQ,Zhang ZH, et al. Activation of microglia in paraventricular nucleus of hypothalarnus :A source of central cytokine release in heart failure[J].Circulation,2005,112(17) : 192.
  • 2Kakimura J , Kitamura Y, Takata K, et al. Microglial activation and amyloid beta clearance induced by exogenous heat - shock proteins[J]. FASEB, 2002,16(6) :601 - 603.
  • 3Acarin L, Gonzalez B, Castellano B, et al. Microglial response to Nmethyl Daspartate mediated excitotoxicity in the immature rat brain [J].Comp Neurol, 1996,367:361 - 374.
  • 4Smita TV, Elkabes S, Schick C, et al. Gene expession in activated brain microglia: Identification of a proteinase inhibitor that increase microglial cell number[J].Mol Brain Res, 1998, 56: 99- 107.
  • 5Paris D, Town T, Mullan M. Novel strategies for opposingmurine microglial activation[J ]. Neurosci Lett, 2000,278 (122) : 528.
  • 6Fiebich BL, Butcher RD. Protein kinase C mediated regulation of NOS expression in cultured microglia [J]. Neuroimmunol, 1998, 92 (122): 170- 178.
  • 7Nagai A, Nakagawa E, Hatori K, et al. Generation and characterization of immortalized human microglial cell lines: Expression of cytokines and chemokines [ J ]. Neurobiol Dis, 2001,8 ( 6 ) : 1057 - 1068.
  • 8汪长胜,霍正禄,杨瑞和.脑缺血后胶质细胞分子改变及相互作用[J].国外医学(脑血管疾病分册),2001,9(1):11-13. 被引量:5
  • 9Park YK,Chung YS, Kim YS. Inhibition of gene expression and production of iNOS and TNF - alpha in LPS - stimulated microglia by methanol extract of Phellodendri cortex [ J ]. Int Immunopharmacol,2007, 7(7): 955-962.
  • 10Millward JM, Caruso M, Campbell IL. IFN - { gamma } induced chemokines synergize with pertussis toxin to promote T cell entry to the central nervous system[J].Immunol, 2007, 178 (12) : 8175 - 8182.

二级参考文献19

  • 1Napper GA, Pianta MJ, Kalloniatis M. Reduced glutamate uptake by retinal glial cells under ischemic/hypoxic conditions. Vis Neurosci,1999,16( 1 ): 149-158.
  • 2Westenbroek RE, Bausch SB, Lin RC, et al. Upregulation of L-type Ca2+ channels in reactive astrocytes after brain injury, hypomyelination,and ischemia. J Neurosci, 1998,18(7) :2321-2334.
  • 3Gottlieb M, Matute C. Expression of ionotropic glutamate receptor subunits in glial cells of the hippocampal CA1 area following transient forebrain ischemia. J Cereb Blood Flow Metab, 1997,17(3) :290-300.
  • 4Cotrina ML, Kang J, Lin JH, et al. Astrocytic gap junctions remain open during ischemic conditions. J Neurosci, 1998,18(7) :2520-2537.
  • 5Li WE, Ochalski PA, Hertzberg EL. Immunorecognition, ultrastructure and phosphorylation status of astrocytic gap junctions and connexin43 in rat brain after cerebral focal ischaemia. Eur J Neurosci, 1998, 10(7):2444-2463.
  • 6Verkhratsky A, Kettenmann H. Calcium signalling in glial cells. Trends Neurosci, 1996,19(8) :346-352.
  • 7Irving EA, Yatsushiro K, McCulloch J. Rapid alteration of tau in oligodendrocytes after focal ischemic injury in the rat: involvement of free radicals. J Cereb Blood Flow Metab, 1997,17(6):612-622.
  • 8Ludwin SK. The pathobiology of the oligodendrocyte. J Neuropathol Exp Neurol, 1997,56(2): 111-124.
  • 9Yamashita K, Sakurai-Yamashita Y, Niwa M, et al. The glial endothelin-nitric oxide system in ischemia-related neuronal cell death. Nippon Yakurigaku Zasshi, 1998,111 ( 1 ) :29-36.
  • 10Nakashima MN, Yamashita K, Kataoka Y, et al. Time course of nitric oxide synthase activity in neuronal, glial, and endothelial cells of rat striatum following focal cerebral ischemia. Cell Mol Neurobiol, 1995,15(3) :341-349.

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