1Ogawa M, Hirota M, Hayakawa T, et al. Development and use of a new staging system for severe aeute panereatisis based on a nationwede suevey in Japan [J]. Panereas, 2002,25 (4) : 325- 330.
2Steer ML. Relationship between pancreatitis and lung disease[J]. Respir Physiol,2001,128(1) :13-16.
3Siebenlist U, Franzoso C, Brown K, et al. Structure, regulation and function of NF-kappa B[J]. Annu Rev Cell Biol, 1994,10: 405-455.
4Yamazaki S, Muta T, Takeshige K. A novel IkappaB protein, Ikap-paB-zeta,induced by proinflammatory stimuli,negatively regulates nuelearfaetor-kappaB in the nuelei[J]. J Biol Chem, 2001,276(29) :27657-27662.
5Gray KD, Simovic MO, Chapman WC, et al. Systemic nfkappaB activation in a transgenic mouse model of acute pancreatitis[J]. J Surg Res,2003,110(1):310-314.
6Fujita M, Masamune A, Satoh A, et al. Ascites of rat experimental model of severe acute pancreatitis induces lung injury[J]. Pancreas,2001,22(4) : 409-418.
7Jaffray C, Yang J, Carter G, et al. Pancreatic elastase activates pulmonary nuclear factor kappa B and inhibitory kappa B, mimicking pancreatitis-associated adult respiratory distress syndrome [J]. Surgery,2000,128(2) :225-231.
8Geisler F, Algul H, Riemann M, et al. Questioning current concepts in acute pancreatitis: endotoxin contamination of porcine pancreatic elastase is responsible for experimental pancreatitis-associated distant organ failure [J]. J Immunol, 2005,174(10) :6431-6439.
9Algul H, Trelber M, Lesina M, et al. Pancreas-specific RelA/ p65 truncation increases susceptibility of acini to inflammation-associated cell death following cerulein pancreatitis[J]. J Clin Invest, 2007,117(6): 1490-1501.
10Inoue S,Nakao A,Kishimoto W, et al. LFA-1 (CD11a/CD18) and ICAM-1 (CD54) antibodies attenuate superoxide anion release from polymorphonuclear leukocytes in rats with experimental acute pancreatitis[J]. Pancreas,1996,12(2) :183- 188.
1Ryter SW,Kim HP, Nakahira K, et al. Protective functions ofheme oxygenase-1 and carbon monoxide in the respiratorysystem[J], Antioxid Redox Signal,2009 ,9(12) :2157-2173.
2Lundvig DM, Immenschuh S, Wagener FA. Heme oxygenase,inflammation, and fibrosis: the good,the bad, and the ugly.[J]. Front Pharmacol, 2012 3 : 81. doi: 10 3389/fphar. 2012.00081.
3Jianbo Y,Shuan D,Xiaoqing L,et al. Role of HO-1 inprotective effect of electro-acupuncture against endotoxinshock-induced acute lung injury in rabbits[J]. Exp Biol Med(Maywood),2013,238(6) : 705-712.
4Jin Y,Choi AM. Cytoprotection of Heme Oxygenase-1/Carbon Monoxide in Lung Injury [J]. Proc Am Thorac Soc,2005,2(3):232-235.
5Poss KD,Tonegawa S. Reduced stress defense in hemeoxygenase 1-deficient cells[J]. Proc Natl Acad Sci USA,1997 ,94(20):10925-10930.
6Lee PJ, Alam J, Wiegand GW,et al. Overexpression of hemeoxygenase-1 in human pulmonary epithelial cells results in cellgrowth arrest and increased resistance to hyperoxia[J]. ProcNatl Acad Sci USA, 1996,93(19) : 10393-10398.
7Xie W, Wang H, Wang L, et al. Resolvin D1 reducesdeterioration of tight junction proteins by upregulating HO-1in LPS-induced mice[J]. Lab Invest,2013,93(9) :991-1000.
8Sheu CC, Zhai R, Wang Z,et al. Hemeoxygenase-1microsatellite polymorphism and haplotypes are Associatedwith the development of acute respiratory distress syndrome[J], Intensive Care Med,2009,35(8) : 1343-1351.
9Li J, Li D, Liu X. et al. Human umbilical cord mesenchymalstem cells reduce systemic inflammation and attenuate LPS-induced acute lung injury in rats[J]. J Inflamm (Lond) ,2012,9(1):33.
10Demedts M, Costabel U. ATS/ERS internationalmultidisciplinary consensus classification of the idiopathicinterstitial pneumonias [J]. Eur Respir J,2002, 19 (5): 794-796.
