期刊文献+

应激性溃疡发病机理研究近展 被引量:5

下载PDF
导出
摘要 应激性溃疡是各种应激因素导致的急性胃粘膜病变,其发病机制包括中枢神经系统和神经内分泌传导机制紊乱引起的局部H^+浓度增加、血液循环障碍和胃粘膜屏障功能破坏。体液因素如前列腺素、去甲肾上腺素、乙酰胆碱、组织胶等含量的变化可改善或加重胃粘膜病变。近年来认为,超氧离子、某些神经肽类物质亦可通过某种机制参与应激性溃疡的形成。
出处 《国外医学(消化系疾病分册)》 CAS 1990年第4期228-231,共4页
  • 相关文献

参考文献8

  • 1Hiroyuki Yoshimura PhD,Noboru Kan MD,Nobuya Ogawa MD. Preventive and curative effects of prostaglandins on stress ulcer in rats[J] 1989,Digestive Diseases and Sciences(3):436~444
  • 2Koji Takeuchi PhD,Youichi Nobuhara PhD,Hironori Tanaka BS,Hideyuki Nishiwaki BS,Susumu Okabe PhD. Effects of hemorrhagic shock on alkaline secretion and mucosal tolerance to acid in rat duodenum[J] 1988,Digestive Diseases and Sciences(11):1409~1417
  • 3Stanislaw J. Konturek MD,Tomasz Brzozowski PhD,Danuta Drozdowicz MSc,Gerhard Beck PhD. Role of leukotrienes in acute gastric lesions induced by ethanol, taurocholate, aspirin, platelet-activating factor and stress in rats[J] 1988,Digestive Diseases and Sciences(7):806~813
  • 4Nicola Basso MD,Maurizio Bagarani MD,A. Eugene Pekary MD,Alfredo Genco MD,Alberto Materia MD. Role of thyrotropin-releasing hormone in stress ulcer formation in the rat[J] 1988,Digestive Diseases and Sciences(7):819~823
  • 5C. Ritter MD,R. A. Hinder FRCS, PhD,M. M. J. Oosthuizen PhD,L. G. Svensson FRCS, PhD,S. J. S. Hunter BSc (HONS),H. Lambrecht BSc. Gastric mucosal lesions induced by hemorrhagic shock in baboons[J] 1988,Digestive Diseases and Sciences(7):857~864
  • 6David Wiles,Oktay Yeginsu MD,Ali Ozden MD,Steve M. Covington MD,Janet Rice PhD,Atilla Ertan MD. Gastric, duodenal, and pancreatic somatostatin-like immunoreactivity during hypovolemic shock[J] 1988,Digestive Diseases and Sciences(7):878~882
  • 7Dr. Tsuyoshi Yabana MD, PhD,Akira Yachi MD, PhD. Stress-induced vascular damage and ulcer[J] 1988,Digestive Diseases and Sciences(6):751~761
  • 8Koji Takeuchi PhD,Susumu Okabe PhD. Importance of pepsin and stomach distension in morphological alterations of stress-induced gastric lesions in pylorus-ligated rats[J] 1988,Digestive Diseases and Sciences(1):52~59

同被引文献21

引证文献5

二级引证文献11

相关作者

内容加载中请稍等...

相关机构

内容加载中请稍等...

相关主题

内容加载中请稍等...

浏览历史

内容加载中请稍等...
;
使用帮助 返回顶部