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B类清道夫受体CD36与动脉粥样硬化研究进展 被引量：2

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摘要 B类清道夫受体(SR)CD36是一个分布广泛,生物作用多样的多配基受体,参与体内多种病理、生理过程。在动脉粥样硬化(AS)形成过程中其介导单核细胞与内皮细胞黏附,是单核-巨噬细胞识别、内吞ox-LDL的主要SR,参与泡沫细胞和AS形成。CD36表达的调控机制一直是研究的热点,特别是PPARγ的作用,而相关药物如临床上广泛应用的作为胰岛素增敏剂的PPARγ激动剂以及他汀类降脂药等对CD36表达的影响也引起人们的关注。
作者 孙颖 常志文
机构地区 首都医科大学附属北京同仁医院干部医疗科
出处 《心脏杂志》 CAS 2008年第2期232-236,共5页 Chinese Heart Journal
关键词 清道夫受体 CD36 动脉粥样硬化
分类号 R541.4 [医药卫生—心血管疾病]
  • 相关文献

参考文献19

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二级参考文献36

共引文献20

同被引文献24

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  • 3董晓秋,王璐璐,毕伟,金钟男.卵巢巧克力囊肿无水乙醇硬化治疗前后卵巢功能的变化[J].中国超声医学杂志,2007,23(12):939-942. 被引量：12
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  • 10史精华,冷金花,宋楠,董喆,郎景和.腹腔镜卵巢子宫内膜异位囊肿剥除术对卵巢储备功能的影响[J].现代妇产科进展,2010,19(7):481-485. 被引量：47

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心脏杂志
2008年 第2期

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