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Ⅳ型胶原酶与糖尿病肾病 被引量:2

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出处 《泰山医学院学报》 CAS 2007年第10期837-840,共4页 Journal of Taishan Medical College
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  • 1黄翠玲,李才,邓义斌,王丽娟,张秀云,赵丽艳.大黄对糖尿病大鼠肾组织非酶促糖基化的影响[J].中国糖尿病杂志,1996,4(2):103-106. 被引量:56
  • 2Fornoni A, Wang Y C, Lenz O, et al. Association of a decreased number of d(CA) repeats in the matrix metalloproteinase-9 promoter with glomerulosclerosis susceptibility in mice. J Am Soc Nephrol, 2002,13:2068-2076.
  • 3Naito T, Razzaque MS, Nazneen A, et al. Renal expression of the Ets-1 proto-oncogene during progression of rat crescentic glomerulonephritis. J Am Soc Nephrol, 2000,11:2243-2255.
  • 4Asanuma K, Shirato I, Ishidoh K, et al. Selective modulation of the secretion of proteinases and their inhibitors by growth factors in cultured differentiated podocytes. Kidney Int, 2002,62:822-831.
  • 5Noel LH. Morphological features of primary focal and segmental glomerulosclerosis. Nephrol Dial Transplant,1999,14( Suppl 3):53-57.
  • 6McMillan J I, Riordan J W, Couser W G, et al. Characterization of a glomerular epithelial cell metalloproteinase as matrix metalloproteinase-9 with enhanced expression in a model of membranous nephropathy. J Clin Invest,1996,97:1094-1101.
  • 7Urushihara M, Kagami S, Kuhara T, et al.Glomerular distribution and gelatinolytic activity of matrix metalloproteinases in human glomerulonephritis. Nephrol Dial Transplant, 2002,17:1189-1196.
  • 8Mundel P, Reiser J, Borja AZM ,et al. Rearrangements of the cytoskeleton and cell contacts induce process formation during differentiation of conditionally immortalized mouse podocyte cell lines. Exp Cell Res,1997, 236:248-258.
  • 9Kriz W, Gretz N, Lemley K V. Progression of glomerular diseases: is the podocyte the culprit? Kidney Int, 1998,54:687-697.
  • 10Basile DP. The transforming growth factor beta sytem in kidney disease and repair: recent progress and future directions. Curr Opin Nephrol Hypertens, 1999,8:21-30.

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