1Saito T. Refractory nephrotic syndrome[J]. Nippon Rinsho, 2004,62(10) : 1794.
2Hess J, Angel P, Schorpp-Kistner M. AP-1 subunits: quarrel and harmony among siblings[J]. J Cell Science, 2004,117 (25) : 5965.
3Karin M,Chang L. AP-l-glucocorticoid receptor crosstalk taken to a higher level [J]. J Endocrinol, 2001,169 (3) : 447.
4Vats A, Nayak A, Ellis D, et al. Familial nephritic syndrome: clinical spectrum and linkage to chromosome 19q13[J]. Kidney Int, 2000,57 : 875.
5Ruf RG, Lichtenberger A, Karle SM, et al. Patients with mutations in NPHS2 (podoein)do not respond to standard steroid treatment of nephrotie syndrome[J]. J Am Soc Nephrol, 2004,15 : 722.
6Kim JM, Wu H, Green G, et al. CD2-associated protein haploinsufficiency is linked to glomerular disease susceptibility [J]. Science, 2003,300 : 1298.
7Kaplan JM, Kim SH, North KN, et al. Mutations in ACTN4 ,encoding alpha-actinin-4 cause familial focal segmental glomerulosclerosis[J]. Nat Genet,2000,424 - 251.
8Kusaba T,Konno Y, Hatta S, et al. More stable and relia- ble pharmacokinetics with preprandial administration of cyclosporine compared with postprandial administration in patients with refractory nephrotic syndrome[J]. Pharmacotherapy, 2005,25 (1) : 52.
9Shiiki H, Saito T, Nishitani Y, et al. Prognosis and risk factors for idiopathic membranous nephropathy with nephrotic syndrome in Japan[J]. Kidney Int, 2004,65 (4):1400.
10Muso E, Mune M, Yorioka N, et al. Beneficial effect of low-density lipoprotein apheresis (LDL-A) on refractory nephrotic syndrome (NS) due to focal glomerulosclerosis (FGS)[J]. Clin Nephrol,2007,67(6) :341.
