摘要
低钠血症是急性出血性脑卒中较为常见的并发症之一,严重影响患者的预后。根据其发生机制主要有2种综合征:抗利尿激素分泌异常综合征(SIADH)和脑耗盐综合征(CSWS),临床上SIADH和CSWS的治疗方案截然相反,只有了解患者发生低钠血症的病理生理学机制,才能有效地纠正低钠血症,并防止可能出现的并发症。现介绍急性出血性脑卒中出现低钠血症患者的临床表现、诊断、发生机制及治疗方面的研究进展。
Hyponatremia is one of more common complications in acute cerebral hemorrhagic stroke and influences patients prognosis. There are two syndromes about its mechanism : syndrome of inappropriate antidiuretic hormone secretion (SIADH) and cerebral salt wasting syndrome (CSWS). Owing to contrary therapeutic plan between SIADH and CSWS, we can improve effectively hyponatremia and prevent latent complications only by pathophysiological mechanism of hyponatremia in patients with acute cerebral hemorrhagic stroke. This article introduced the clinical performance, diagnosis, treatment and mechanism of patients with acute cerebral hemorrhagic stroke and hyponatremia.
出处
《医学综述》
2008年第9期1347-1349,共3页
Medical Recapitulate
关键词
急性出血性脑卒中
低钠血症
抗利尿激素分泌异常综合征
脑耗盐综合征
Acute cerebral hemorrhagic stroke
Hyponatremia
Syndrome of inappropriate antidiuretic hormone secrection
Cerebral salt wasting syndrome