1Rutkowski DT, Kaufman RJ. A trip to the ER: coping with stress. Trends Cell Biol, 2004,14: 20-28.
2Feng B. The endoplasmic reticulum is the site of cholesterol-induced cytotoxicity in macrophages. Nat Cell Biol, 2003, 5: 781- 792.
3Reddy RK. Maom C, Baumeister P, et al. Endoplasmic reticulum chaperone protein GRP78 protects cells from apoptosis induced by topoisomerase inhibitors: role of ATP binding site in suppression of caspase-7 activation. J Biol Chem, 2003, 278: 20915-20924.
4Ellgaard L, Helenius A. Quality control in the endoplasmic reticulure. Nat Rev Mol Cell Biol, 2003, 4: 181-191.
5Rao RV, Hermel E, Castro-Obregon S, et al. Coupling endoplasmic reticulum stress to the cell death program. Mechanism of caspase activation. J Biol Chem, 2001, 276: 33869-33874.
6Nakagawa T, Zhu H, Morishima N, et al. Caspase-12 mediates endoplasmic-reticulum-specific apoptosis and cytotoxicity by amyloid-beta. Nature, 2000, 403: 98-103.
7Kalai M, Lamkanfi M, Denecker G, et al. Regulation of the expression and processing of caspase-12. J Cell Biol, 2003,162: 457- 467.
8Jimbo A, Fujita E, Kouroku Y, et al. ER stress induces caspase-8 activation, stimulating cytochrome C release and caspase-9 activation. Exp Cell Res, 2003, 283:156-166.
9Oyadomari S, Mori M. Roles of CHOP/GADD153 in endoplasmic reticulum stress. Cell Death Differ, 2004,11:381-389.
10Tardif KD, Waris G, Siddiqui A. Hepatitis C virus, ER stress, and oxidative stress. Trends Mierobiol, 2005, 13 : 159-162.
10Liver Failure and Artificial Liver Group, Chinese Society of Infectious Diseases, Chinese Medical Association.肝衰竭诊疗指南[J].中华肝脏病杂志,2006,14(9):643-646. 被引量：829
