1William E. Boden. Radomized trial of percutaneous coronary intervention added to optimal medical therapy in patients with stable coronary heart disease:results of the clinical 56^th Annul scientificsession of ACC [J]. Neworlans, 2007,3 : 24-27
2Sarunic MV, Applegate BE, Izatt JA, et al. Spectral domain second-harmonic optical coherence tomography [J]. Opt Lett, 2005, 30(18):2391-2393.
3Tawakol A, Migrino RQ, Hoffmann U, et al. Noninvasive in vivo measurement of vascular inflammation with F-18 fluorodeoxy-glucose positron emission tomography [J]. J Nucl Cardio, 2005, 12 (3):294-301.
5Kolodgie FD,Burke AP,Farb A, et al. The thin-cap fibroatheroma:a type of vulnerable plaque: the major precursor lesion to a cute coronary syndromes [J]. Curr Opin Cardiol, 2001,16 (5) : 285- 292.
6Kitselaer B,Dogma J,Narula J. Non-invasive detection of plaque instability using radiolabeled Annexin-V in patients with atherosclerotic carotid artery disease[J]. N Engl J Med ,2004,350 (9):1472-1473.
二级参考文献15
1[1]Shak PK. Role of inflammation and metalloproteinases in disruption and thombosis. Vas Med, 1998,3 (3): 199-206
2[2]Ihling C. Pathomorphology of coronary atherosclerosis. HERZ, 1998, 23 (2):69-77
3[3]Van der Wal AC, Becker AE,Van der Loos CM,et al. Site of intimal rupture or erosion of thrombosed coronary atherosclerotic plaques is characterized by an inflammartory process irrespectlve of the dominant plaque morphoRLogy. Circulation, 1994,89 ( 1 ) :36-44
4[4]Kaartinen M, Van der Wal AC, Van der Loos CM, et al. Mast cell infiltration in acute coronary syndromes: implications for plaque rupture. J Am Coll Cardial, 1998,32(3) :606-612
5[5]Mehta JL, Saldeen TG, Rand K. Interactive role of infection, inflammation and traditional risk factors in atherosclerosis qand coronary artery disease. J Am Coll Cardiol, 1998, 31 (6): 1217-1225
6[6]Rebuzzi AG, Quaranta L, Liuzzo G, et al. Incremental prognostic value of serum levels of tropinin T and C-reactive protein on admission in patients with unstable angina pectoris. Am J Cardiol, 1998,82: 715-719
7[7]Verheggen PWHM, De Maat MPM, Manger Cats V, et al. Inflammation status as a main determinant of outcome in patients with unstable angina, independent of coagulation activation and endothelial cell function. Eur Heart J, 1999,20: 567-574
8[8]Ridker PM, Cushman M, Stampfern M J, et al. Inflammation, aspirin and the risk of cardiovascular disease in apparently healthy men. N Engl J Med, 1997,336(14): 973-979
9[9]Futterman LG, Lenberg L. Inflammation in plaque rupture:participant or an invited guest ? Am J Crit Care, 1998,7 (2): 153-161
10[10]Gupta S, Leatham EW, Carrington D, et al. Elevated chlamydia pneumoni ae antibodies, cardiovascular events and azithromycin in male survivors of acute mycardial infarction. Circulation, 1997,96: 404-407