摘要
The aryl hydrocarbon receptor （AhR） was discovered almost 30 years ago as a specific binding site for the halogenated polycyclic aromatic hydrocarbon, 2,3,7,8-tetrachlorodibenzo-p-dioxin （TCDD）, an environmental toxin （as reviewed in [1]）. Within the last decade, AhR was found to have a basic helixloop-helix and function as a ligand-activated transcription factor.
