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脑缺血性损伤中细胞凋亡的研究进展 被引量:6

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作者 杨彦玲
出处 《临床神经病学杂志》 CAS 北大核心 2008年第3期236-237,共2页 Journal of Clinical Neurology
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参考文献20

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二级参考文献45

  • 1李小冰,刘岳文,郅兴义.Caspase家族与心脏移植细胞凋亡[J].河南科技大学学报(医学版),2004,22(1):76-78. 被引量:3
  • 2Sobey CG. Cerebrovascular dysfunction after subarachnoid haemorrhage: novel mechanisms and directions for therapy[J].Clin Exp Pharmacol Physiol, 2001, 28(11):926.
  • 3Sun BL, Xia ZL, Yang MF, et al. Effects of Ginkgo biloba extract on somatosensory evokedpotential, nitric oxide levels in serum and brain tissue in rats with cerebral vasospasm after subarachnoid hemorrhage[J]. Clin Hemorheol Microcirc, 2000, 23(2-4):139.
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  • 6Gursoy-Ozdem ir Y, Bolay H, Saribas O, et al. Role of endothelial nitric oxide generation and peroxynitrite formation in reperfusion injury after focal cerebral ischemia[J]. Stroke, 2001, 31(8):1974.
  • 7Robertson GS, Crocker SJ, Nicholson DW, et al. Neuroprotection by the inhibition of apoptosis[J]. Brain Pathol, 2000, 10(2):283.
  • 8Smaili SS,Hsu YT,Youle RJ,et al.Mitochondria in Ca^2+ signaling and apoptosis.Bioenerg Biomember,2000,32(1):35-37.
  • 9Kenichi I,Michael D,Charles S,et al.Transgenic expression of Bcl-2 modulates energy metabolism ,prevents cytosolic acidification during ischemia,and reduces ischemia/reperfusion injury.Circ Res,2004,1(10):734-741.
  • 10Stacey MO,Rosemary AK,Frederick AM,et al.Ischemia preconditioning protects against gut dysfunction and mucosal injury after ischemia/reperfusion injury.Shock,2005,23(3):258-263.

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二级引证文献43

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