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肝硬化组织转化生长因子β_1(TGFβ_1)的表达及临床意义 被引量:2

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作者 蒋莹 赵金仙
出处 《新疆医科大学学报》 CAS 2008年第6期739-739,共1页 Journal of Xinjiang Medical University
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  • 1Zweig M H,Clin Chem,1993年,39卷,561页
  • 2Bauer M, Schuppan D. TGFbetal in liver fibrosis: time to change paradigms? FEBS Lett. 2001,502(1-2) :1-3.
  • 3Basile AS,Gammal SH,Jones EA, et al. GABAA receptor complex in experimental model of hepatic encephalopathy: evidence for elevated levels of an endogenous bezodiazepine receptor ligand. J Neurochem, 1989, 53 ( 4 ):1057-1063.
  • 4Weiner FR, Giambrone MA, Czaja MJ, et al. Ito-cell gene expression and collagen regulation . Hepatology, 1990,11(1):111-117.
  • 5Charlotte F, Win KM, Preaux AM, et al . Immunolocalization of Heparinbinding of growth factors (HBGF) type 1 and 2 in rats liver. Selective hyperexpression of HBGF-2 in carbon tetrathloride-induced fibrosis. J Pathol,1993,169(4) :471-476.
  • 6Yu C, Wang F, Jin C,et al. Role of fibroblast growth factor type 1 and2 in carbon tetrarchlovide-induced hepatic injury and fibrogenesis. Am J Pathol,2003,163(4): 1653-1662.
  • 7Jones SG,Morrisey K,Williams JD,et al. TGF-betal stimulates the.release of pre-formed bFGF from renal proximal tubular cells. Kidney Int, 1999,56(1) : 83-91.

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