大鼠缺血性卒中光化学模型研究

Research on photochemical ischemic stroke model in rat

本文基于光化学反应机理，对大鼠缺血性卒中光化学模型进行研究，指出光化学反应诱导ＶＥＣ功能异常，从而加速了血小板聚集，导致血管栓塞。该模型制作简单，相对非侵害，有可能替代大脑动脉闭塞的缺血性卒中模型。

On the basis of photochemical reaction mechanism,the ischemic stroke photochemical model is studied and indicates that photochemical reaction accelerates platelet aggregation by induced vascular endothelial cells(VEC)dysfunction resulted in vascular embolism.The model is made simple,relative non invasive and may replace ischemic stroke model of cerebral atrery embolism.