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酮色林对HepG2细胞Sp1表达的影响

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出处 《肝脏》 2008年第4期327-328,共2页 Chinese Hepatology
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  • 1张冲 张金山 黄威权 等.5-羟色胺及其受体与人肝细胞性肝癌增殖的关系[J].中华肝脏病杂志,2000,6:345-345.
  • 2Wang S, Wang W, Wesley RA, et al . A Spl binding site of the tumor necrosis factor promoter functions as a nitric oxide response element. J Biol Chem, 1999, 274:33190-33193.
  • 3Borgatti M, Boyd DD, Lampronti I, et al. Decoy molecules based on PNA DNA chimeras and targeting Spl transcription factors inhibit the activity of urokinase-type plasminogen activator receptor (uPAR) promoter. Oncol Res, 2005, 15: 373-383.
  • 4de Wolf CJ, Cupers RM, Bertina RM, et al. The constitutive expression of anticoagulant protein S is regulated through multiple binding sites for Spl and Sp3 transcription factors in the protein S gene promoter. J Biolchem, 2006, 281: 17635-17643.
  • 5Sugawara H, Iwata H, Ichinose A. Regulation of human protein Z gene expression by liver enriched transcription factor HNF-4alpha and ubiquitous factor Sp1. J Thromb Haemost, 2007,5:250-2258.
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  • 7Huang XF, Zhang YQ, Bu YA, et al. Vasoactive intestinal peptide stimulates the growth of hepotoma cell in vitro. Clin Med Sci J, 1993,8:147-150.

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