过氧化物酶体增殖物激活受体与有氧运动能力研究进展
出处
《中国运动医学杂志》
CAS
CSCD
北大核心
2008年第5期651-654,共4页
Chinese Journal of Sports Medicine
基金
国家自然科学基金(项目编号:30570898)资助
参考文献25
-
1Issemaan I, Green S. Activation of a member of the steroid hormone receptor superfamily by peroxisome proliferators.Nature, 1990, 347--645.
-
2Wang YX, Zhang CL, Yu RT, et al. Regulation of muscle fiber type and running endurance by PPARdelta. PLoS Biol, 2004,2 (10):e294.
-
3Guan Y & Breyer MD. Peroxisome proliferator--activated receptors (PPARs):novel therapeutic targets in renal disease. Kidney Int, 2001,60:14--30.
-
4Guan Y, Zhang Y, Breyer D, et al. The role of PPARs in the transcriptional control of cellular processes. Drug News and Perspective, 2002,15 : 147-- 154.
-
5Feige JN, Gelman L, Michalik L, et al. From molecular action to physiological outputs: peroxisome proliferatoractivated receptors are nuclear receptors at the crossroads of key cellular functions. Prog Lipid Res, 2006, 45: 120--159.
-
6Jones S, Savory R, Barratt P, et al. Chromosoma localization, imducibility, tissue-specific expression and strain differences in three murine perioxisome-proliferator-aetivated-receptor genes. Eur J Biochem, 1995, 233:219--226.
-
7Braissant O, Foufelle F, Scotto C, et al. Differential expression of peroxisome proliferator-activated receptors (PPARs) : tissue distribution of PPAR-alpha, -beta, and -gamma in the adult rat. Endocrinology, 1996, 137(1):354 --366.
-
8Oliver J, Shenk J, Snaith R. et al. A selective peroxisome proliferator-activated receptor-delta agonist promotes reverse cholesterol transport. Proe Natl Acad Sci USA,:003, 98: 5306--5311.
-
9Terada S, Wicke S, Holloszy JO, et al. PPARdelta activator GW-501516 has no acute effect on glucose transport in skeletal muscle. Am J Physiol Endocrinol Metab, 2006,290: E607--11.
-
10Schoonjans K, Peinado-Onsurbe J, Lefebvre AM, et al. PPARα and PPARγ activators direct a distinct tissue-specific transcriptional response via a PPRE in the lipoprotein lipasegene. EMBOJ, 1996, 15(19): 5336--5348.
-
1张玲玉,叶鹏,Marvar PJ,Thabet SR,Guzik TJ,Lob HE,McCann LA,Weyand C,Gordon FJ,Harrison DG.血管紧张素Ⅱ诱导的高血压所引起的T淋巴细胞活化与血管炎症的中枢和外周机制[J].中华高血压杂志,2010,18(12):1172-1172. 被引量:15
-
2曲宝戈,王相茂.慢性心力衰竭时运动耐量变化的外周机制及其可逆性[J].国外医学(物理医学与康复学分册),1995,15(1):7-10. 被引量:5
-
3彭志伟,雷雨.低氧防治肥胖的可能性探讨[J].中国科技博览,2009(14):144-145.
-
4池肇春.肠易激综合征发病机制进展[J].中国医师进修杂志,2013,36(16):1-3. 被引量:2
-
5王磊,潘化平,高真真,王尊.二甲双胍对糖耐量异常患者有氧运动能力及运动过程中能量代谢的影响[J].中国康复医学杂志,2016,31(3):324-327. 被引量:10
-
6王向东,刘荣华,马宝娟,董立书.内脏脂肪含量对青年肥胖女性心肺功能及有氧运动能力的影响[J].现代预防医学,2010,37(10):1907-1909. 被引量:10
-
7罗斌钰(译者),赵咏桔(审校者),刘建民(审校者).生长激素替代治疗改善甲状腺素生物学效应:在治疗中枢性甲状腺功能减退中的意义[J].中华内分泌代谢杂志,2008,24(1):107-108. 被引量:1
-
8郑华,原晓晶.康复训练对冠心病患者有氧运动功能的影响[J].中华物理医学与康复杂志,2012,34(4):293-294. 被引量:18
-
9许瑛.日本宇都宫济生会病院心脏康复护理见闻[J].南方护理学报,2005,12(6):89-90. 被引量:6
-
10孙宇楠,段琼玉,吴荣.PPARγ抗肺纤维化机制的研究进展[J].实用肿瘤学杂志,2015,29(1):73-77. 被引量:4