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强直性脊柱炎发病机制的研究进展 被引量:7

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作者 王斌 王萍萍
出处 《四川医学》 CAS 2008年第10期1429-1431,共3页 Sichuan Medical Journal
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  • 1Khan MA, Mathieu A, Sorrentino R, et al. The pathogenetie role of HLA-B27 and its subtypes[J] .Autoimmun Rev,2007,6(3): 183 - 189
  • 2Smith JA, Marker-Hermann E, Colbert RA. Pathogenesis of ankylosing spondy-litis : Current concepts[J]. Best Pract Res Clin Rheumatol, 2006,20 (3) :571 - 591
  • 3Lai E, Teodoro T, Volchuk A. Endoplasmic reticulum stress: signaling the unfolded protein response[ J]. Physiology(Betesda), 2007,22:193 - 201
  • 4Lopez de Castro JA. HLA-B27 and the pathogenesis of spondyloarthropathies [ J ]. Immunol Lett, 2007,108 ( 1 ) : 27 - 33
  • 5May E, Dorris ML, Satumtira hi, et al. CD8 alpha beta t cells are not essential to the pathogenesis of arthritis or colitis in HLA-B27 transgenic rats [J]. J Immunol, 2003,170(2) : 1099 - 1105
  • 6Tran TM, Satumtira N, Dorris ML, et al. HLA-B27 in transgenic rats forms disulfide-linked heavy chain oligomers and multimers that bind to the chaperone BiP[J] .J Immunol,2004, 172(8) :5110 - 5119
  • 7Saleki K, Hartigan N, Lith M, et al. Differential oxidation of HLA-B2704 and HLA-B2705 in lymphoblastoid and transfected adherent cells[J]. Antioxid Redox Signal ,2006,8(3 - 4) :292 - 299
  • 8Turner MJ, Sowders DP, DeLay ML, et al. HLA-B27 misfolding in transgenic rats is associated with activation of the unfolded protein response[J]. J Immunol,2005,175(4) :2438 - 2448
  • 9Galocha B, de Castro JA. Folding of HLA-B27 subtypes is determined by the global effect of polymorphic residues and shows incomplete correspondence to ankylosing spondylitis [ J ]. Arthritis Rheum, 2008, .58 ( 2 ) : 401 - 412
  • 10Stone MA, Payne U, Sehentag C, et al. Comparative immune responses to candidate arthritogenic bacteria do not confirm a dominant role for Klebsiella pneumonia in the pathogenesis of familial ankylosing spondylitis[J]. Rheumatology(Oxford) ,2004,43(2) : 148 - 155

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