脑梗死后磁共振短T_1信号的影像学表现

MRI Manifestation of Short T_1-signal after Cerebral Ischemia Infarction

目的通过回顾204例脑梗死患者的MR影像分析脑梗死后短T1信号的分布、表现特点,对其可能的机制进行的分析和推测。方法分析我院204例脑梗死患者的MR成像资料,均经临床及影像学证实。所有病例常规行SET1WI、FSET2WI、PDWI。结果204例脑梗死患者中102例出现短T1信号,102例未出现短T1信号。102例脑梗死后短T1信号病例123个短T1信号灶分布3个区,皮质区占60%,皮质下区占30%,基底核区占10%;表现为3种形态:(1)线状,占49%;(2)斑点状,占13%;(3)斑片状,占38%。与脑梗死病灶的病灶的关系有3种:(1)皮质面,占56%;(2)病灶范围内,占34%;(3)白质面,占10%。结论缺血性脑梗死后短T1信号不仅在皮质区出现,还可在皮质下区和基底核区出现。缺血性脑梗死后短T1信号的病理生理学机制包括:再灌注损伤;选择性的神经元损伤。

Objective This study investigates the radiological feature of short - T1 signal after cerebral ischemia infarction, and preliminarly analyse and predict the mechenism of the short - T1 signal. Methods To respectively analyse MRI feature of 204 patients of cerebral ischemia infarction identified by clingic and imaging. All of cases was performed conventionally SE T1WI, FSE T2WI, PDWI. Results 102 of 204 patients of cerebral ischemia infarction appear short - T1 signal on MRI. 123 lesions with short -T1 signal in 102 patients distribute in 3 type regions, include cortical region 60% （n =74）, subcortical region 30% （n=37）, basal ganglia region 10% （n = 12）. It demonstrated 3 form, include linar 49% （n =60）, punctate 13% （n = 37）, pathy 38% （ n = 47）. There is 3 type relationship with infarction include cortical surface 56% （ n = 69）, centre of infarction lesion 34% （ n = 42） , White matter around 9% （ n = 11 ） , it major located margin around the lesion. Conclusion Short -T1 signal after cerebral ischemia infarction not only demonstrate in cortical region, but also demonstrate in subcortical and basal ganglia regions. The potential pathophysiology precesses of short - T1 signal after cerebral ischemia infarction includes reperfusion- induced injury, sective nouren necrosis.