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细胞周期依赖性蛋白激酶5在学习和记忆中的作用

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摘要 细胞周期依赖性蛋白激酶5(Cdk5)通过与突触前、突触后物质及下游效应因子之间的相互作用参与学习、记忆过程。病理状态下,Cdk5被其激活因子p25过度激活,参与缺血性脑损伤和神经变性病的发生,因此降低Cdk5异常增高的活性有可能成为改善学习和记忆的新途径。
出处 《国际神经病学神经外科学杂志》 2008年第5期458-462,共5页 Journal of International Neurology and Neurosurgery
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