1Hug H. Fas-mediated apoptosis intumor and defense. Biol Chem,1997,378(12): 1405
2Ohmori T, Podack ER, Nishio K, et al. Apoptosis of lung cancer cells caused by someanti-cancer agents (MMC, CPT-11, and ADM) is inhibited by Bcl-2. Biochem Bioph Res Commun,1993,1921 (1): 30
3Oltavai ZN,Millimen CL,Korsmeyer SJ,et al. Bcl-2 heterodimerizes in vivo withconserved homolog, Bax, that accelerates programmed cell death. Cell, 1993,74 (4): 609
4Boise LH, Gonzalez-Garcia M, Postema CE, et al. Bcl-x, a Bcl-2 related gene thatfunctions as dominant regulator of apoptotic cell death. Cell, 1993,74 (3): 597
5Yang E,Zha JP,Jockel J ,et al. Bad,a heterodimeric partner for BclXL and Bcl-2,displaces Bax and promotes cell death. Cell, 1995,80 (2):285
6Takayama S, Sato T, Krajewski S, et al. Cloning and functional analysis of BAG-1: anovel Bcl-2 binding protein with anti-cell death activity. Cell, 1995,80 (2): 279
7Reed JC. Bcl-2 and the regulation of programmed cell death. J Cell Bid, 1993,75(2):241
9Vescio R,Berenson J. Lichtenstein aupregulated expression of Bcl-2inmutiplemyltiple myeloma cells induced by exposure to doxorubicin etoposideand hydrogenperoxide. Blood, 1996,88(5): 1805
10Naumann U, Weller M. Retroviral Bax gene transfer fails to sensitize malignantglioma cell to CD95L-induced apoptosis and cancer chemotherapy. Iht J Cancer, 1998,77(4):645