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CTLA-4基因多态性与Graves病 被引量:2

Cytotoxic T Lymphocyte Associated Antigen 4 gene Polymorphisms and Graves' disease
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出处 《解剖与临床》 2008年第6期456-458,共3页 Anatomy and Clinics
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  • 1Gough SC. The genetics of Graves disease. Endocrinol Metab Clin North Am,2000,29(2):255-266.
  • 2Polymeropoulos MH, Xiao H, Rath DS, et al. Dinucleotide repeat polymorphism at the human CTLA-4 gene. Nucleic Acids Res, 1991,19(14):4018.
  • 3Kouki T, Gardine CA, Yanagawa T, et al. Relation of three polymorphisms of the CTLA-4 gene in patients with Graves disease. J Endocrinol Invest, 2002,25(3): 208-213.
  • 4Hadj Kacem H, Bellassoued M, Bougacha-Elleuch N, et al. CTLA-4 gene polymorphisms in Tunisian patients with Graves' disease. J Clin Immunol, 2001, 101(3):361-365.
  • 5Kotsa K, Watson PF, Weetman AP. A CTLA-4 gene polymorphisms is associated with both Graves disease and autoimmune hypothyroidism. Clin Endocrinol, 1997,46(5):551-554.
  • 6Dariavach P, Mattei MG, Golstein P, et al. Human Ig superfamily CTLA-4 gene: chromosomal localization and identity of protein sequence between murine and human CTLA-4 cytoplasmic domains. Eur J Immunol, 1998,18(12):1901.
  • 7Heward JM, Allahabadia A, Armitage M, et al. The development of Graves' disease and the CTLA4 gene on chromosome 2q^33. J Clin Endocrinol Metab,1999, 84(7):2398-2401.
  • 8Tomer Y, Greenberg DA, Barbesino G, et al. CTLA4 and not CD28 is a susceptibility gene for thyroid autoantibody production. J Clin Endocrinol Metab,2001,86(4): 1687-1693.
  • 9Kinjo Y,Takasu N, Komiya I, et al. Remision of Graves' hyperthyroidism and A/G polymorphism at position 49 in exon 1 of cytotoxic T lymphocyte-associated molecule-4 gene. Clin Endocrinol Metab,2002,87(6):2593-2596.
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  • 1杨建军,张毓洪,霍正浩,朱小泉,宋亮,杨泽.CTLA-4基因的多态性与宁夏儿童1型糖尿病的相关性研究[J].宁夏医学院学报,2006,28(5):374-378. 被引量:8
  • 2刘英,刘巍,李为民,李悦,孔一慧,甘润韬,王政,樊瑛,耿建强,邵群,张美,高成,王秀荣.CTLA-4基因多态性与特发性扩张型心肌病的相关性[J].中国免疫学杂志,2007,23(1):66-69. 被引量:2
  • 3余永胜,汤正好,冯洁,奚敏,张毅,臧国庆.慢性乙肝患者外周血T淋巴细胞CTLA4表达及意义[J].中国临床医学,2007,14(4):493-495. 被引量:3
  • 4Varpula M, Pulkki K, Karlsson S, et al. Predictive value of N-ter- minal pro-brain natriuretic peptide in severe sepsis and septic shock [J]. Crit Care Med, 2007,35(5):1277-1283.
  • 5Waterhoues P, Penninger JM, Timms E, et al. Lymphoproliferative disorders with early lethality in mice deficient in Ctla-d [ J ]. Sci- ence, 1995,270 (5238) :985-988.
  • 6Li HS, Ligons DL, Rose NR. Genetic complexity of autoimmune myocarditis [ J ]. Autoimmunity Rev, 2008,7 ( 3 ) : 168-173.
  • 7Hagiya M, Francarilla A, Polimeno L, et al. Cloning and sequence analysis of the rat augmenter of liver regeneration (ALR) gene: ex- pression of biologically active reeomblnation ALR and demonstration of tissue distribution[J]. Proc Natl Aead Sci USA, 1994,91 (17) : 8142-8146.
  • 8Firestein GS. Evolving concepts of rheumatoid arthritis[ J ]. Nature, 2003,423 (6937) : 356-361.
  • 9Kristiansen OP, Larsen ZM, Pociot F. CTLA4 in autoimmune dis- ease-a general susceptibility gene to autoimmunity [ J ]. Genes im- mun, 2000,1(3) :170-184.
  • 10Varpula M,Pulkki K,Karlsson S,et al. Predictive value of N-terminal pro brain natriuretic peptide in severe sepsis and septic shock[J]. Crit Care Meal,2007,35(5) : 1 277-1 283.

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