胫骨平台后内侧骨折的创伤机制及生物力学研究

The biomechanical research of postermedial tibial plateau fracture

目的:研究胫骨平台后内侧骨折创伤机制的生物力学意义。方法:选用成人离体膝关节标本,在屈曲90°的情况下给予加载速率为40 mm·min^(-1)的垂直载荷,绘制时间-载荷曲线,记录造成后内侧骨折的最大载荷及伴随损伤情况。结果:在膝关节屈曲90°予以轴向载荷可致胫骨平台后内侧骨折,且多伴前交叉韧带损伤。结论:膝关节屈曲90°轴向载荷是导致胫骨平台后内侧骨折的一种创伤机制,ACL损伤在此创伤机制中占有重要作用。

Objective ： To study the biomechanical significance of traumatic mechanism of postermedial tibial plateau fracture. Methods： The models were produced by adult cadaver knee joints, apeak stress to models was given when it was in flexion 90°, the velocity was 40 mm· min^-1 The time - stress curve was protracted and the furthest stress that caused the tibial plateau fracture and concomitancy trauma was registered. Results： The postermedial tibial plateau fracture was caused when apeak stress was given to knee joint in flexion 90°, combined with anterior cruciate ligament damaged. Conclusion： The stress of axial direction on the knee joint in flexion 90° is a traumatic mechanism of postermedial tibial plateau fracture, and anterior cruciate ligament injuries plays an important role in this mechanism.