超短波对多形核白细胞释放β-葡萄糖醛酸酶的影响

Effect of ultrashort wave on βglucuronidase resulting from polymorphonuclear

目的：从生物分子学水平研究超短波的作用机制。方法：将大鼠分成正常组、炎症组、活体组、试管组，观察大鼠体液在超短波作用前后多形核白细胞（ＰＭＮｓ）释放β－葡萄糖醛酸酶（β－ｇ）的情况。结果：正常大鼠的体液不论在活体内，还是在试管内，超短波作用后β－ｇ大量释放；当大鼠有炎症时，酶大量释放的情况下，超短波可抑制酶的释放。结论：超短波对ＰＭＮｓ释放β－ｇ有双向调节作用。

Objective:This study was to investigate the mechanism of ultrashort wave to βglucuronidase of rats at the level of molecular biology.Methods:Rats were divided into normal group and inflammation group.After ultrashort wave radiation,the level of βglucuronidase resulting from polymorphonuclear(PMN) was studied both in vivo and in vitro.Results:The release of βglucuronidase from PMN by ultrashort wave radiation was greatly increased both in vivo or in vitro in normal rats.When the rats were infected with inflammation,the enzyme release was inhibited by ultrashort wave radiation.Conclusion:It is shown that the ultrashort wave has dual regulatory effects for βglucuronidase release.