1Thomas DB, Franceschini N, Hogan SL, et al. Clinical and pathologic characteristics of focal segmental glomerulosclerosis pathologic variants. Kidney Int, 2006, 69:920-926.
2D'Agati VD, Fogo AB, Bruijn JA, et al. Pathologic classification of focal segmental glomerulosclerosis: a working proposal. Am J Kidney Dis, 2004, 43:368 - 382.
3Petermann A, Floege J. Podocyte damage resulting in podocyturia: a potential diagnostic marker to assess glomerular disease activity. Nephron Clin Pract, 2007, 106 :c61 - c66.
4Friedrich C, Endlich N, Kriz W, et al. Podocytes are sensitive to fluid shear stress in vitro. Am J Physiol Renal Physiot, 2006, 291 : F856 - 17865.
5Bariety J, Mandet C, Hill GS, et al. Parietal podocytes in human glomeruli. J Am Soc Nephrol, 2006, 17:2770 - 2780.
6Sagrinati C, Netti GS, Mazzinghi B, et al. Isolation and characterization of muhipotent progenitor cells from the Bowman's capsule of adult human kidneys. J Am Soc Nephrol, 2006, 17:2443 -2456.
7Becker JU, Hoerning A, Schmid KW, et al. Immigrating progenitor cells contribute to human podocyte turnover. Kidney Int, 2007, 72: 1468 - 1473.
8Stokes MB, Valeri AM, Markowitz GS, et al. Cellular focal segmental glomerulosclerosis: clinical and pathologic features. Kidney Int 2006, 70 : 1783 - 1792.
9Eremina V, Baelde HJ, Quaggin SE. Role of the VEGF a signaling pathway in the glomerulus : evidence for crosstalk between components of the glomerular filtration barrier. Nephron Physiol, 2007, 106 : 32 - 37.
10Lai KW, Wei CL, Tan LK, et al. Overexpression of interleukin-13 induces minimal change-like nephropathy in rats. J Am Soe Nephrol, 2007, 18 : 1476 - 1485.
9Qian Y, Feldman E, Pennathur S, et al. From fibros is to scleros is mechanisms of glomerulo seleros is in diabetic nephropathy. Diabetes ,2008,57 ( 6 ) : 1439.
10Vaziri ND, Navab M, Fogelman AM. HDL metabolism andactivity in chronic kidney di.sease. Nat Rev Nephro1,2010,6 (5) :287 - 296.
