摘要
Ten cases of gastrointestinal adenocarcinomas were histochemically investigated fortheir lectin receptors by avidin—biotin-peroxidase complex(ABC)method.Results showed thatgastric adenocarcinomas expressed more types of lectin receptors than large boweladenocarcinomas.Positive rates of Glc/Man-and GlcNAc-specific lectin receptors in these tu-mors were kigher than those of Gal/GalNAc-and Fuc-specific lectin receptors.From the stom-ach to rectum,the spectrum of the lectin receptors in pericarcinomatous tissues graduallychanged from the gastric type to the rectal type.The expression of lectin receptors ingustrointestinal adenocarcinomas was obviously different Jrom that in their correspondingpericarcinomatous tissues.The increase and decrease of lectin receptor expression,theappearance and disappearance of some types of lectin receptors as well as the change of thereceptor distribution are indirectly regulated by cellular genome.
Ten cases of gastrointestinal adenocarcinomas were histochemically investigated for their lectin receptors by avidin—biotin-peroxidase complex(ABC)method.Results showed that gastric adenocarcinomas expressed more types of lectin receptors than large bowel adenocarcinomas.Positive rates of Glc/Man-and GlcNAc-specific lectin receptors in these tu- mors were kigher than those of Gal/GalNAc-and Fuc-specific lectin receptors.From the stom- ach to rectum,the spectrum of the lectin receptors in pericarcinomatous tissues gradually changed from the gastric type to the rectal type.The expression of lectin receptors in gustrointestinal adenocarcinomas was obviously different Jrom that in their corresponding pericarcinomatous tissues.The increase and decrease of lectin receptor expression,the appearance and disappearance of some types of lectin receptors as well as the change of the receptor distribution are indirectly regulated by cellular genome.
