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肥胖与多囊卵巢综合征关系 被引量:5

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出处 《医学研究杂志》 2009年第4期127-128,133,共3页 Journal of Medical Research
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参考文献12

  • 1Apridonidze T,Essah P, Luorno M, et al. Prevalence and characteristics of the metabolic syndrome in women with polycystic ovary syndrome [ J ]. Clin Endocrinol Metab, 2005 ,90 : 1929 - 1935
  • 2徐谷根,李萍,罗婉宜,叶仁青,蔡德鸿.葡萄糖耐量减低患者体重指数、腰围/臀围比与血压、血脂的关系[J].实用医学杂志,2008,24(11):1917-1919. 被引量:3
  • 3曹云霞,魏兆莲.肥胖与多囊卵巢综合征[J].中国实用妇科与产科杂志,2004,20(12):709-711. 被引量:16
  • 4胡敏,侯丽辉,吴效科.瘦素与多囊卵巢综合征的研究进展[J].中国妇幼健康研究,2006,17(2):125-128. 被引量:9
  • 5鲁晓燕,刘曼华.多囊卵巢综合征的高雄激素血症[J].南通大学学报(医学版),2007,27(2):154-156. 被引量:4
  • 6Crosignani PG, Colombo M , Vegetti W, et al. Overweight and obese anovulatory patients with polycystic ovaries : parallel improvements in anthropometric indices , ovarian physiology and fertility rate induced by diet[J]. Hum Reprod ,2003 ,18 (9) :1928-1932
  • 7Pasquali R, Pelusi C, Genghini S, Caceeiari M, Gambineri A. Obsity and reproductive disorders in Women [ J ]. Hum Reprod Update,2003, 9:359 - 372
  • 8Creaswell JL, Barker D J, Osmond C, et al. Fetal growth,length of gestation, and polycystic ovaries in adult life [ J ]. Lancet, 1997,350 : 1131 - 1135
  • 9Eisner JR, Dumesic DA, Kemnitz JW, Abbott dh. Timing of prenatal androgen excess determines differential impairment in insulin secretion and action in adult female rhesus monkeys [ J ]. Clin Endocrinal Metab ,2000,85 : 1206 - 1210
  • 10Abbott DH, Dumesic DA,Franks S. Developmental origin of polycystic ovary syndrome - a hypothesis[ J ]. Endocrinal,2002,174 : 1 - 5

二级参考文献67

  • 1陈坚梅,顾维正.肥胖与糖尿病[J].医师进修杂志,2004,27(8):11-13. 被引量:8
  • 2[1]Azziz R.PCOS:A diagnostic challenge[J].Reprod Biomed Online,2004,8(6):644-648.
  • 3[2]Timar O,Sestier F,Levy E.Metabolic syndrome X[J].Can J Cardiol,2000,16(6):779-789.
  • 4[3]Lee CY,Baik KH,Parkb C,et al.Relationships of plasma insulin-like growth factor (IGF)-Ⅰ and IGF-Ⅰ I concentrations to growth in purebred Landrace and Yorkshire female pigs[J].Liv Prod Sci,2005,95(1-2):163-169.
  • 5[4]San Millan JL,Corton M,Villuendas G,et al.Association of the polycystic ovary syndrome with genomic variants related to insulin resistance,type 2 diabetes mellitus,and obesity[J].J Clin Endocrinol Metab,2004,89(6):2640-2646.
  • 6[5]Sesfi G,Federiei M,Hribal ML,et al.Defects of the insulin receptor substrate (IRS) system in human metabolic disorders[J].FASEB J,2001,15(12):2099-2111.
  • 7[6]Yen HW,Jakimiuk AJ,Munir I,et al.Selective alterations in insulin receptor substrates-1,-2 and -4 in theca but not granulose cells from polycystic ovaries[J].Mol Hum Reprod,2004,10(7):473-479.
  • 8[7]Rice S,Christoforidis N,Gadd C,et al.Impaired insulin-dependent glucose metabolism in granulose-lutein cells from anovulatory women with polycystic ovaries[J].Hum Reprod.2005,20(2):373-381.
  • 9[8]Witchel SF,Smith R,Tomboc M,et al.Candidategeneanalysis in premature pubarche and adolescent hyperandrogenism[J],Fertil Steril,2001,75(4):724-730.
  • 10[9]Moran C,Reyna R,Boots LS,et al.Adrenocortical hyperresponsiveness to corticotrophin in polycystic ovary syndrome patients with adrenal androgen excess[J].Fertil Steril,2004,81:126-131.

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引证文献5

二级引证文献62

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