期刊文献+

溃疡性结肠炎免疫学机制的研究进展 被引量:5

原文传递
导出
作者 闫聿逊
出处 《临床消化病杂志》 2009年第2期126-128,共3页 Chinese Journal of Clinical Gastroenterology
  • 相关文献

参考文献17

  • 1IVAN JF, FRANK H, MONICA B, et al. Nonclassical CDld-restricted NF T ceils that produce IL- 13 characterize an atypical Th2 response in ulcerative colitis[ J]. J Clin Invest,2004,113:1490.
  • 2HELLER F,FLORIAN P, BOJARSKI C, et al. Interleukin-13 is the key effector Th2 cytoking in ulcerative colitis that affects epithelial tight junctions, apoptosis, and cell restitution [ J ]. Gastroenterology, 2005,129:550.
  • 3ELIZABETH F, TOSEI M, KLAUS YM, et al. lmmunopathogenesis of eXperimental ulcerative colitis is mediated by eosinophil peroxidase [ J ]. J Immunol,2004,172:5664.
  • 4RANDALL HG, FIONA MT , ALAN GB, et al. Deficiency of invariant NK T cells in Crohn' s disease and ulcerative colitis[ J]. Dig Dis Sci ,2007,52 : 1415.
  • 5YOKO Y,KEN F, YOSHIHIRO F, et al. Demonstration of low-regulatory CD 25 high + CD 4 + and high-pro-inflammatory CD 28 - CD 4 + T-cell subests in patients with ulcerative colitis:modified by selective granulocyte and monocyte adsorption apheresis [ J ]. Dig DisSei,2007,52:2725.
  • 6STURM A,LEITE AZA, DANESE S, KRIVACIC KA,et al. Divergent cell cycle kinetics underlie the distinct functional capacity of mucosal T cells in Crohn' s disease and ulcerative colitis[ J]. Gut, 2004,53 : 1624.
  • 7AMEZCUA-GUERRA LM, HERNANDEZ-MARTINEZ B, PINEDA C. Gut,2006,55 : 1059.
  • 8TAKAHASHI M, NAKAMURA K, HONDA K, et al. An inverse correlation of human peripheral blood regulatory T cell freguency with the disease activity of ulcerative colitis [ J ]. Dig Dis Sci, 2006,51 : 677.
  • 9PEPPELENBOSCH MP P,VAN DEVENTER SJH. T cell apoptosis and inflammatory bowl disease[J]. Gut,2004,53:1556.
  • 10KOUTROVBAKIS IE, DRYQIANNAKJS D, KARMIRIS K, et al. Pancreatic antoantibodies in greek patients with inflammatory bowel disease [ J]. Dig Dis Sci ,2005,50:2330.

同被引文献80

引证文献5

二级引证文献14

相关作者

内容加载中请稍等...

相关机构

内容加载中请稍等...

相关主题

内容加载中请稍等...

浏览历史

内容加载中请稍等...
;
使用帮助 返回顶部