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自身免疫性甲状腺疾病中Thl/Th2细胞因子的偏移

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出处 《内科》 2009年第2期295-297,共3页 Internal Medicine
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  • 1Elenkov IJ. Glucocorticoids and the Th1/Th2 balance[J]. Ann N Y Acad Sci,2004,1024:138-146
  • 2Kidd P. Th1/Th2 balance: the hypothesis, its limitations, and implications for health and disease[J].Altern Med Rev,2003,8(3):223-246
  • 3Barrett K, Liakata E, Rao PV, et al. Induction of hyperthyroidism in mice by intradermal immunization with DNA encoding the thyrotropin receptor[J]. Clin Exp Immunol,2004,136(3):413-422
  • 4Weetman AP. Cellular immune responses in autoimmune thyroid disease[J].Clin Endocrinol,2004,61(4):405-413
  • 5Rosloniec EF, Latham K, Guedez YB,et al. Paradoxical roles of IFN-gamma in models of Th1-mediated autoimmunity[J]. Arthritis Res,2002,4(6):333-336
  • 6Yu S, Sharp GC, Braley-Mullen H. Dual roles for IFN-gamma, but not for IL-4, in spontaneous autoimmune thyroiditis in NOD.H-2h4 mice[J]. J Immunol,2002,169(7):3999-4007
  • 7Vasu C, Dogan RN, Holterman MJ, et al. Selective induction of dendritic cells using granulocyte macrophage-colony stimulating factor, but not fms-like tyrosine kinase receptor 3-ligand, activates thyroglobulin-specific CD4+/CD25+ T cells and suppresses experimental autoimmune thyroiditis[J]. J Immunol,2003,170(11):5511-5522

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