6Selhub J ,Jacques PF, Wilson PWF, et al. Vitamins status and intake as primary deteriminaants of homocysteinemia in an elderly population. JAMA, 1993,270(22) :2 693 -2 698.
7Ueland PM, Refsum H, Stabler SP, et al. Total homocysteine in plasma of serum:Methods and clinical applications. Clin Chem,1993,39(9) :1 764 - 1 769.
8Boers CHJ. Hyperhomocysteinemia as a rick fator arteria and venous disease a review of evidence and relevance. Trom And Haemos, 1997, 78(1) :580.
2[1]Selhub J, Jacques PF, Rosenberg IH, et al. Serum total homocysteine concentrations in the Third National Health and Nutrition Examination Survey ( 1991-1994 ): population reference ranges and contribution of vitamin status to high serum concertrations. Ann Intern Med , 1999, 131: 331-339.
3[2]Ubbink JB, Becker PJ, Vermaak WJH, et al. Results of B-vitamin supplementation study used in a prediction model to define a reference range for plasma homocysteine. Clin Chem , 1995, 41 (7): 1033-1037.
4[3]Laet CD, Wautrecht JC, Brasseur D, et al. Plasma homocysteine concentrations in a Belgian school-age population. Am J Clin Nutr , 1999, 69: 968-972.
5[4]Verhoef P, Meleady R, Daly LE, et al.Homocysteine, vitamin status and risk of vascular disease. Effects of gender and menopausal status. Eur Heart J, 1999, 20: 1234-1244.
6[5]Herrmann W, Quast S, Ullrich M, et al.Hyperhomocysteinemia in high-aged subjects: relation of B-vitamins, folic acid, renal function and the methylenetetrahydrofolate reductase mutation Atherosclerosis , 1999, 144: 91-101.
7[6]Ubbink JB, Vermaak WJH, Delport R, et al.Effective homocysteine metabolism may protect South African blacks against coronary heart disease. Am J ClinNutr, 1995, 62: 802-808.
8[7]Malinow MR, Ducimetiere P, Luc G, et al. Plasma homocysteine levels and graded risk for myocardial infarction: findings in two populations at contrasting risk for coronary heart disease. Atherosclerosis ,1996, 126: 27-34.
9[8]Alfthan G, Pekkanen J, Jauhiainen M, et al. Relation of serum homosysteine and lipoprotein ( a )concentrations to atherosclerotic disease in a prospective Finnish population based study.Atherosclerosis, 1994, 106 (1): 9-19.
10[9]Girelli D, Friso S, Trabetti E, et al.Methylenetetrahydrofolate reductase C677T mutation,plasma homocysteine, and folate in subjects from Northern Italy with of without angiographically documented severe coronary atherosclerotic disease:Evidence for an important genetic-environmental interaction. Blood, 1998, 91: 4158-4163.
