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破骨细胞分化发育中的信号转导及转录因子研究进展 被引量:3

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出处 《山东医药》 CAS 北大核心 2009年第19期111-113,共3页 Shandong Medical Journal
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参考文献8

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二级参考文献15

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共引文献18

同被引文献48

  • 1孟祥阁.破骨细胞分化的信号转导系统[J].科协论坛(下半月),2009(1):75-75. 被引量:1
  • 2文剑明,郑铭豪.破骨细胞的形成、功能及细胞因子的调节[J].中国骨质疏松杂志,1998,4(1):64-68. 被引量:8
  • 3吴鹰,沈志强.破骨细胞空泡型质子泵及其抑制剂的研究进展[J].国外医学(药学分册),2005,32(3):188-192. 被引量:1
  • 4Niilura K.vacuolar ATPase as a drug discovery. Drug News Perspect.2006; 19(3): 139-144.
  • 5Cipriano DJ,Wang Y,Bond 5,et al.Structure and regulation of the vacuolar ATPase. Biochim Biophys Acta. 2008; 1777(7-8): 599-604.
  • 6Lacey DL,Timms E,Tan HL,et al.Osteoprotegerin ligand is a cytokine that regulates osteoclast differentiation and activation. Cell.2008;93(2): 165-176.
  • 7Nordstron T, Shrode LD,Rotstein OD,et aI.Chronic extracellular acidosis induces plasmalemmal vacuolar type H^+ ATPase activity in osteoclasts.J Biol Chem.2007;272: 6354-6360.
  • 8Beutler JA,McKee TC.Novel marine and microbial natural product inhibitors of vacuolar ATPase.Curr Med Chem. 2003 10(9):787-796.
  • 9Zhao Q,Shao J,Chen W,et aI.Osteoclast differentiation and gene regulation. Front Biosci.2007; 12:2519-2529.
  • 10Li YP, Chen W,Liang Y, et al. Atp6i deficient mice exhibit severe osteopetrosis due to loss of osteoclast-me-diated extracellular acidification.Nat genet.2009; 23(4):447-451.

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二级引证文献16

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